Getbig Misc Discussion Boards > Religious Debates & Threads
Jihadists shouting "Allahu akbar" murder 13 customs agents
Colossus_500:
So you're going to "refute" the fact that chronic marijuana use does not erode the gray matter (hippocamus, not sure if this is the correct spelling or terminology)? Give me a minute, I'm sure I can google up a study real quick. You lose all credibility with me if you even remotely disagree with this.
Colossus_500:
Here's an article that sums up my argument of erosion of the gray matter portion of the brain:
www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=2576305&dopt=Abstract
Colossus_500:
Here's another article (note where this came from - you should already know this being the genious that you are):
Chronic Cannabis Abuse Raises Nerve Growth Factor Serum Concentrations in Drug-Naive Schizophrenic Patients
Maria C. Jockers-ScherüBl
maria.jockers@medizin.fu-berlin.de
Uta Matthies
Heidi Danker-Hopfe
Undine E. Lang
Richard Mahlberg
Rainer Hellweg
Department of Psychiatry and Psychotherapy, Charite-University Medicine Berlin, Campus Benjamin Franklin, Berlin, Germany
Long-term cannabis abuse may increase the risk of schizophrenia. Nerve growth factor (NGF) is a pleiotropic neurotrophic protein that is implicated in development, protection and regeneration of NFG-sensitive neurones. We tested the hypothesis that damage to neuronal cells in schizophrenia is precipitated by the consumption of cannabis and other neurotoxic substances, resulting in raised NGF serum concentrations and a younger age for disease onset. The NGF serum levels of 109 consecutive drug-naive schizophrenic patients were measured and compared with those of healthy controls. The results were correlated with the long-term intake of cannabis and other illegal drugs. Mean (± SD) NGF serum levels of 61 control persons (33.1 ± 31.0 pg/ml) and 76 schizophrenics who did not consume illegal drugs (26.3 ± 19.5 pg/ml) did not differ significantly. Schizophrenic patients with regular cannabis intake (> 0.5 g on average per day for at least 2 years) had significantly raised NGF serum levels of 412.9 ± 288.4 pg/ml (n = 21) compared to controls and schizophrenic patients not consuming cannabis (p < 0.001). In schizophrenic patients who abused not only cannabis, but also additional substances, NGF concentrations were as high as 2336.2 ± 1711.4 pg/ml (n = 12). On average, heavy cannabis consumers suffered their first episode of schizophrenia 3.5 years (n = 21) earlier than schizophrenic patients who abstained from cannabis. These results indicate that cannabis is a possible risk factor for the development of schizophrenia. This might be reflected in the raised NGF-serum concentrations when both schizophrenia and long-term cannabis abuse prevail.
Johnny Apollo:
--- Quote from: Colossus_500 on April 13, 2006, 11:07:37 AM ---Here's an article that sums up my argument of erosion of the gray matter portion of the brain:
www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=2576305&dopt=Abstract
--- End quote ---
Try READING the study before posting it...
--- Quote ---Test results did not suggest that chronic THC exposure produced behavioral deficits resembling those seen following hippocampal damage. Habituation rates in an activity monitor were identical for all exposure groups, and there was no passive avoidance retention deficit. Further, while TMT caused pronounced abnormalities in the complex maze, chronic THC exposure at the two highest dose levels significantly improved maze performance, similar to BZ effects on this task.
--- End quote ---
Exposure to THC(Chemical in marijuana) IMPROVED maze performance!
::)
Johnny Apollo:
--- Quote from: Colossus_500 on April 13, 2006, 11:15:56 AM ---Here's another article (note where this came from - you should already know this being the genious that you are):
Chronic Cannabis Abuse Raises Nerve Growth Factor Serum Concentrations in Drug-Naive Schizophrenic Patients
Maria C. Jockers-ScherüBl
maria.jockers@medizin.fu-berlin.de
Uta Matthies
Heidi Danker-Hopfe
Undine E. Lang
Richard Mahlberg
Rainer Hellweg
Department of Psychiatry and Psychotherapy, Charite-University Medicine Berlin, Campus Benjamin Franklin, Berlin, Germany
Long-term cannabis abuse may increase the risk of schizophrenia. Nerve growth factor (NGF) is a pleiotropic neurotrophic protein that is implicated in development, protection and regeneration of NFG-sensitive neurones. We tested the hypothesis that damage to neuronal cells in schizophrenia is precipitated by the consumption of cannabis and other neurotoxic substances, resulting in raised NGF serum concentrations and a younger age for disease onset. The NGF serum levels of 109 consecutive drug-naive schizophrenic patients were measured and compared with those of healthy controls. The results were correlated with the long-term intake of cannabis and other illegal drugs. Mean (± SD) NGF serum levels of 61 control persons (33.1 ± 31.0 pg/ml) and 76 schizophrenics who did not consume illegal drugs (26.3 ± 19.5 pg/ml) did not differ significantly. Schizophrenic patients with regular cannabis intake (> 0.5 g on average per day for at least 2 years) had significantly raised NGF serum levels of 412.9 ± 288.4 pg/ml (n = 21) compared to controls and schizophrenic patients not consuming cannabis (p < 0.001). In schizophrenic patients who abused not only cannabis, but also additional substances, NGF concentrations were as high as 2336.2 ± 1711.4 pg/ml (n = 12). On average, heavy cannabis consumers suffered their first episode of schizophrenia 3.5 years (n = 21) earlier than schizophrenic patients who abstained from cannabis. These results indicate that cannabis is a possible risk factor for the development of schizophrenia. This might be reflected in the raised NGF-serum concentrations when both schizophrenia and long-term cannabis abuse prevail.
--- End quote ---
This study doesn't set out to prove cannabis causes schizophrenia just that users who will already get schizophrenia suffer a relaps earlier than those who don't use.
The study is flawed in many ways.
For instance Correlation does not indicate causation. Just because they happened to use marijuana prior to having schizophrenia does not indicate marijuana CAUSED it. Another likely explanation is the fact that people with schizophrenia are more likely to use it than those without it.
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