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1  Getbig Main Boards / Gossip & Opinions / Re: I have 1 bitcoin on: December 27, 2013, 12:56:40 AM
Thank you. I feel that Bitcoin will eventually be challenged and perhaps even overcome by a challenger. Which one I don't know but it will have to take advantage of Bitcoin's shortcomings. That'll be a good one to get in early on. I couldn't tell you which one of the dozens it could be. I'll be buying some quark coin also as of right now, that's my guess. It's only .12 right now. Alot of positive news on it.

I would go with Litecoins as the second most established alternative.

The main issue is making traceable refunds, if one of these currencies can do this reliably thats the one to invest in.

Apparently only 250,000 people in the world own more than one bitcoin.
2  Getbig Main Boards / Gossip & Opinions / Re: RIP Scott Peckham on: December 27, 2013, 12:54:21 AM
RIP Scott.

From a friend --> "Not sure what happened, but more info will be coming soon. One of his close friends is saying it was a heart attack and it happened around midnight Monday. One of his family members also stated that it was a heart attack at the gym after training"



sad, after intense exercise, this is one of the times when steroid users are at the highest risk of having a heart attack.
3  Getbig Main Boards / Gossip & Opinions / Re: The city I live in on: December 21, 2013, 05:55:18 AM
get some of this business

4  Getbig Main Boards / Gossip & Opinions / Re: Guam on: December 21, 2013, 03:09:01 AM

aviation
5  Getbig Main Boards / Gossip & Opinions / Life in Guam? on: December 21, 2013, 01:34:54 AM
Anybody here visited Guam or lived there??

Possibly going out there for work so looking for some feedback...

6  Getbig Main Boards / Gossip & Opinions / Re: The city I live in on: December 21, 2013, 12:56:31 AM
Looks like shit.  Hope this helps

Here is where I live.

You have nice trees, they have better women Grin
7  Getbig Main Boards / Gossip & Opinions / Re: The city I live in on: December 21, 2013, 12:56:00 AM
Here is Galati, Romania. Here's what I currently call home.




Post about the city YOU live in.

Ce Faci Ma?

What is this part of Romania like, I have been along the cost but only from Mamaia to Venus.
8  Getbig Main Boards / Gossip & Opinions / Re: Ruhl is scared to die on: December 15, 2013, 02:37:04 AM
No IGF, no muscle gains though  Angry

For those who want development to the level that only chemical enhancement can give, then yes!
9  Getbig Main Boards / Gossip & Opinions / Re: Ruhl is scared to die on: December 15, 2013, 02:33:36 AM
It should lessen the stress on the heart I'm sure. Though from what I've read the changes to the heart are permanent to a large extent.

But if Ruhl were to get completely off he would be in an extremely weakened state and whatever health issues he has might finish him right off... just complete speculation on my part, just a hunch. Tapering off would be good, but imagine if he's been on daily thyroid, steroids, gh, insulin for a couple of decades straight and stopped all that at once. Sounds dangerous to me, the whole system would go into shock, blood sugar would be all over the place, thyroid all messed up, huge water loss all at once etc. Cheesy

I remember on musclemayhem a decade ago or so when cswole said he got off his bottle a day habit. He was complaining of feeling like utter shit, couldn't sleep for weeks etc. Sounded like a junkie going cold turkey. Cheesy

The changes to the heart are permanent, however massively reducing the workload it has to do by losing weight is going to have a significant benefit. Additionally there are still plenty of things that can be done to maintain and improve heart function, even if it is compromised.
A genuinely healthy diet that is low in calories (not 400 grams of lean protein a day as muscle and fitness would have you believe) would help.
I believe any bodybuilder can sufficiently turn his situation around. Ok, there are some things that cannot change, and there may be some lifelong increased risks that they will have to accept, but many things can be done to reduce those risks.

Unfortunately most bodybuilders both competitive and recreational, are either limited by their intellect, lack of common sense or simply "head in the sand" merchants who cannot make the changes to prevent their early expiration.
10  Getbig Main Boards / Gossip & Opinions / Re: Ruhl is scared to die on: December 15, 2013, 02:27:09 AM
ah yeah good point,lol.

i was thinking for some reason its just the steorids,haha.
he will live on trt or soemthing liek that,will feel very bad till ge gets small.yes iner organs i dont see how they can get smaller in size.
would suprise me if hes off good blood sugar and liver health,i remember his advice for gaining size was the daily proteina nd then top that up with whatever you can fit in the stomach.

i forgot the insulin, t3s , gh.
hell i know t3 levels will recover quickly, if not, t4 for life.

hgh, tbh i dont know if exogonus gh will shut down endogenous production, someone look that up.

insulin, well, that he can stop,if hes diabetic,then its gonna be type 2.

not sure he will ta[per off at all,though.

diabloblanco, yes theres studies theyre very clear eating little is best as far lifespan is ocncerned.

btw im not even sure ruehl is one of the biggest eaters


eating less food and especially less protein has been shown to extend life in various organisms.

By reducing calories and protein you reduce IGF-1, this slows the rate of cell division. In its most basic form the slower the rate of cell division, the longer the cells live. If all your cells collectively divide more slowly you will live longer, assuming you dont get run over by a truck.

All these supplement companies saying take this and that to boost IGF-1, its just accelerating your aging process.
11  Getbig Main Boards / Gossip & Opinions / Re: Boxer Herbie Hide sent to prison on: December 06, 2013, 01:41:31 PM

No surprise.....he was always a complete muppett.
12  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 30, 2013, 03:20:26 AM
I know the exact mechanism by which fibrosis develops following damaged muscle cells. The physiological progression isn't what I am uncertain of.

The question posed by "honest" was:

Whereby, the bolded statement targets the notion of fibrosis as it relates to elasticity in the heart of an individual like that of member "honest" where he changed his lifestyle and training habits from younger days which resulted in reversal of his ventricular wall thickness, but is still wondering as to whether or not steroid use will lead to cardiac muscle fibrosis, in light of positive lifestyle changes being undertaken.

I believe he was looking further into this statement by "lord chronos":

Whereas, my punting over of the question involves not the need for an explanation behind the exact mechanism in which muscle fibrosis occurs after prolonged damage, but, moreover, whether or not any peer-reviewed studies or actual clinical cases that can be pegged, show that lord chronos' statements are in fact justifiable.

Again, the pathology behind fibrosis is very much so understood. My punting over to you was meant in order to see if you have read of any actual studies that can be provided to member "honest" and/or seen clinical cases (during CT surgery whereby heart tissue damage can be directly visualized and/or by witnessed autopsies) that suggest that steroid use led to blatant myocardial fibrosis.

Since I haven't read too many studies surrounding the idea of "Steroids leading to permanent myocardial fibrosis and micro scarring", I was wondering if you had any direct input on the matter based on factual material.

"1"




There are a few studies that discuss the other side effects of steroids such as myocardial fibrosis. I dont have all of the them to hand, as I looked at this about ten years ago and posted a few on a few forums and got the same head in the sand denialist mentality from people who didnt want to discuss.

Fineschi V, Riezzo I, Centini F, et al. Sudden cardiac death during anabolic steroid abuse: morphologic and toxicologic findings in two fatal cases of bodybuilders. Int J Legal Med 2007;121:48–53.

Includes autopsy details with extensive information on scarring and fibrosis in two bodybuilder deaths.

www.researchgate.net/.../79e4150767bf7374d5.pdf‎


Ohio State University review

www.anabolicsteroidcalcu lator.com/resources/articles/.../chapter18.pdf‎

Sudden death is the most frightening consequence of AAS use. The etiology of these events likely is multifactorial, with AAS use contributing to the observed pathology. There are case reports of myocardial infarctions [49], stroke, and peripheral vascular obstruction [50] from thrombus that likely are related to the changes in platelet function, inflammation, and cho- lesterol metabolism discussed above. Autopsies of 34 users of AASs found chronic cardiac changes consisting of cardiac hypertrophy, myocardial fibrosis, and coronary artery atheromatous changes in 12 victims, although these were believed to contribute to the deaths of only 2 victims [48].
Many sudden death events among AAS users have been due to ischemia secondary to coronary artery disease; however, there is a report of ventric- ular tachycardia during exercise testing of an AAS user who had myocardial fibrosis on biopsy [51]. Other case reports of sudden death demonstrate diffuse, patchy fibrotic changes in the myocardium of AAS users without coronary artery atherosclerosis [47]. The presence of scar or infiltrative pro- cesses is commonly believed to be a cause for arrhythmia. The exact cause of sudden death in AAS users is unclear but likely is due to ischemia or arrhythmia.

Cause and manner of death among users of anabolic androgenic steroids.
Thiblin I, Lindquist O, Rajs J.
Source

Department of Forensic Medicine, Karolinska Institute, Stockholm, Sweden.

http://www.sudmed.ru/index.php?act=Attach&type=post&id=4067

Myocardial hypertrophy or other chronic myocardial lesions,
i.e., diffuse or patchy myocardial fibrosis, and myocardial steato-
sis, were noticed in 12 of 25 microscopically investigated cases.

Androgen Abuse and Increased Cardiac Risk John P. Higgins, MD, MPhil, Alireza Heshmat, MD, Christopher L. Higgins, BHMS

http://www.medscape.com/viewarticle/775869_2

Myocardial Histologic Changes

A 2005 study reported two cases of sudden cardiac death in young male athletes related to AAS abuse.[14] Both cases involved healthy individuals without any history of coronary artery disease (CAD) and no evidence of significant abnormality in arterial microscopic examination. Autopsy of both hearts showed focal myocardial fibrosis suggestive of prior myocardial injury. In a study of a sudden unexpected death in a female fitness athlete using steroids and ephedrine, the only pathological finding was a few small foci of granulation tissue, which was interpreted as evidence of earlier myocardial necrosis.[15] Sudden cardiac arrhythmia resulting from inflammatory process and myocardial fibrosis was suggested to be the cause of death in these cases. Other researchers have reported sudden cardiac deaths related to steroids that also showed myocardial fibrosis in the absence of CAD

Reduced Cardiac Function

An echocardiographic study of 47 strength-training individuals (46 male subjects), 28 of whom were regular AAS users, revealed a lower systolic function in AAS users versus nonusers, ejection fractions 58% versus 63%, respectively.[18] In addition, there was evidence of reduced diastolic function by tissue Doppler measurement in the AAS users (ie, their hearts were weaker and stiffer). Another study of 12 long-term AAS users noted that compared with controls, they were noted to have significant systolic cardiac dysfunction as measured by lower left ventricular ejection fraction (50.1% vs 59.1%; P = 0.003), leading the authors to suggest that further work is needed to determine the extent of this problem.[24] An Italian Doppler imaging study also showed reduced systolic function but in a regional distribution.[25]







13  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 30, 2013, 02:43:13 AM
A few things...

If by way of a side effect caused by sheer speculation you happen to learn something about cardiac physiology, that is absolutely fine. That is one of the great things about this forum, in that there is a wealth of information that many members have regarding matters surrounding close to just about anything that is openly shared amongst its members. While sharing information is great, it is very important to make sure that said information is factual and backed by studies and actual clinical data that supports what is being said. If you "learn" something here that is not necessarily true and then share it with others who might be pretty knowledgeable about the matter (say an actual cardiologist etc), it will sound a bit odd to them.

Clearly, there is no suggestion that anyone in this thread is an actual physician. Being a med student does not make one a physician. Heck, being a PGY1-3 (IM resident), also barely makes one a qualified physician since they are still learning and can't practice outside of a hospital.

I think many of the things Danny-boy has said are pretty sound, but there was one thing (the reversibility of cardiac hypertrophy in particular), that I do not believe is correct.

For instance, this statement suggests that:

The statement in bold above is not correct. It is stated as an absolute and it is not necessarily true.

Cardiomyocyte (heart muscle) growth is reversible if caught early on enough. If Danny, or anyone else for that matter, can disprove that ventricular hypertrophy (as seen in patients with HTN-Hypertension) is irreversible, regardless of stage, I'd gladly ask them to post the peer reviewed studies to suggest otherwise.

For instance, here in UpToDate, which tends to be one of the top 5 clinician go-to guides, it states clearly that ventricular hypertrophy (induced growth of cariomyocytes) by way of hypertension can be reversed:



(Notice how under the effects of antihypertensive therapy, it mentions how treatment for HTN will decrease mass in patients with left ventricular hypertrophy and a regression of hypertrophy can be seen. If induced cardiomyocyte growth was truly irreversible, there would be no way for this statement to be true and backed by not just hundreds of studies, but by the American Heart Association)

Not to mention, that it is VERY normal for adamant weightlifters to have mild-moderate left ventricular hypertrophy due to the excessive amount of weights that they lift. Once the stimuli is removed (be it hypertension or prolonged power-lifting, the hypertrophy will typically reverse).

Prolonged hypertrophy that goes untreated can result in heart failure, because like that of a rubber band, the heart muscle can only stretch but so much in order to compensate for the increased workload/demand in putting out oxygenated blood to the systemic circulation.

For more peer-reviewed readings that state clearly that ventricular hypertrophy is reversible, feel free to take a read at:

Ruilope LM, Schmieder RE. Left ventricular hypertrophy and clinical outcomes in hypertensive patients. Am J Hypertens 2008; 21:500.
(http://www.uptodate.com/contents/clinical-implications-and-treatment-of-left-ventricular-hypertrophy-in-hypertension/abstract/20)

Franz IW, Tönnesmann U, Müller JF. Time course of complete normalization of left ventricular hypertrophy during long-term antihypertensive therapy with angiotensin converting enzyme inhibitors. Am J Hypertens 1998; 11:631.
(http://www.uptodate.com/contents/clinical-implications-and-treatment-of-left-ventricular-hypertrophy-in-hypertension/abstract/21)

Cuspidi C, Negri F, Zanchetti A. Angiotensin II receptor blockers and cardiovascular protection: focus on left ventricular hypertrophy regression and atrial fibrillation prevention. Vasc Health Risk Manag 2008; 4:67.
(http://www.uptodate.com/contents/clinical-implications-and-treatment-of-left-ventricular-hypertrophy-in-hypertension/abstract/22)

Okin PM, Devereux RB, Gerdts E, et al. Impact of diabetes mellitus on regression of electrocardiographic left ventricular hypertrophy and the prediction of outcome during antihypertensive therapy: the Losartan Intervention For Endpoint (LIFE) Reduction in Hypertension Study. Circulation 2006; 113:1588. (http://www.uptodate.com/contents/clinical-implications-and-treatment-of-left-ventricular-hypertrophy-in-hypertension/abstract/23)

Os I, Franco V, Kjeldsen SE, et al. Effects of losartan in women with hypertension and left ventricular hypertrophy: results from the Losartan Intervention for Endpoint Reduction in Hypertension Study. Hypertension 2008; 51:1103.
(http://www.uptodate.com/contents/clinical-implications-and-treatment-of-left-ventricular-hypertrophy-in-hypertension/abstract/24)

Solomon SD, Appelbaum E, Manning WJ, et al. Effect of the direct Renin inhibitor aliskiren, the Angiotensin receptor blocker losartan, or both on left ventricular mass in patients with hypertension and left ventricular hypertrophy. Circulation 2009; 119:530.
(http://www.uptodate.com/contents/clinical-implications-and-treatment-of-left-ventricular-hypertrophy-in-hypertension/abstract/25)

That's the only issue I had with Danny's post. The vast majority of what he posted is sound and makes excellent sense, but I disagree in that induced cardiomyocyte growth (as particular seen in ventricular hypertrophy) is irreversible as an absolute statement. If not treated early on, then yes, heart failure can ensue due to excessive cardiac remodeling that after enough time seizes to compensate for the demands of the heart.

If you can post some peer-reviewed studies to substantiate your statements that steroid use will directly lead to the development of myocardial fibrosis and that steroid use is the leading cause of heart failure over other issues like Hypertension, I will gladly read them and evaluate, but for now those are just random, unsubstantiated statements.

Not everything that is spewed should be taken as gospel.

"1"

P.S. I knew Kovacs. I met him through Desmond Miller and Victor Martinez. The guy wasn't the friendliest guy and towards the end (last 5 years of his life) he became pretty bitter towards the "sport".

Hypertrophy is reversible, there is plenty of literature on this, however,  it has also been documented that weight lifting induced hypertrophy in a natural trainer is not the same the hypertrophy in the AAS trainer.
The type of growth is different resulting in slightly different structural results. In the non AAS users, when they stopped training the hypertrophy reduced at a faster rate than the AAS user. Hypertrophy is just one of a myriad of cardiac changes that AAS cause and I would speculate one of the more reversible ones.



14  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 30, 2013, 02:29:51 AM
Once one of us performs his actual biopsy, that's when we will know how prominent the myocardial fibrosis was. Unfortunately, since none of us will get to see what his heart looks like, it's a mute point.

As for now, since no one on this forum will have access to his medical records, all we can do is speculate.

The easiest thing to do is to directly attribute and/or assume that his heart failure was caused due to his steroid use, but without his medical records, past blood work (to see whether he suffered from hyperlipidemia, hypertension, hypo/hyper-thyroidism, diabetes, kidney failure and to even possibly gauge what his BNP was), chest x-ray films, an echocardiogram, ekg, prior stress test results and even a prior CT or MRI scan of his heart, we only have speculation at best. If we had many of these elements, we can then get a better idea of what the full cardiac story was, but going only by pictures off the internet and rumors, we can't possibly assume anything.

To be clear, I'm not saying that steroids could have not played a role, but to directly attribute steroid use to his death is speculation at best.

Lets allow for the man to rest in peace.

"1"

Agreed.

I just find the head in the sand mentality of some AAS users insane. However I think its worthwhile to discuss the points Danny, yourself and few others have raised.
15  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 28, 2013, 03:02:29 PM

I am sure the majority of these bodybuilders were given their fair share of warning signs by their own doctors..yet took that risk.... This lifestyle is an obsessive subculture, where many believe that they are the anomaly and won't succumb to human physiology...  The more realistic bodybuilders ended their careers when they were warned.... I do not doubt that is why you do not see a comeback a la Shawn ray..even though I know he still has the drive to win it all... But am damn sure he now has a precondition like many who were a part of his era..where continuing competitive bodybuilding will have to shorten his lifespan....  Acceptance is a tough pill to swallow....    Nature stopped giving free lunches as soon as they were told..enough is enough...


Agreed. Combination of superior natural genetics, better response to drugs, "moderate use" compared to other less gifted pros, and his success based on presenting an aesthetic physique vs being a mass monster, are for sure all factors why he will be around a lot longer than others.
16  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 28, 2013, 02:59:56 PM
D-B, are you saying there's not free lunch in nature?

Correct
17  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 28, 2013, 02:59:04 PM
On a case by case basis...  hypertrophy of the left ventricle is usually what is seen within these types of patients... where the left ventricle happens to be the corresponding chamber that is ejecting the body's overall systemic blood flow to all your tissues, muscles, organs, etc..... In order to overcome a higher pressure gradient now seen in the aorta...  this left ventricle has to find new avenues in order to compensate and re-establish a new set medium of adequate blood flow and stroke volume that will supply the rest of the body... Further, in order to provide more "force" aka "higher contractility" - more cardiomyocytes need to develop OR hypertrophy may also be seen . Here, the cardiac muscle eventually stiffens and loses its capability to contract adequately... BIGGER heart -> demands greater supply of 02 via the coronary arteries(which directly supply the heart)   quick cardiovascular 101 -> heart is made up of 4 chambers composed of 2 atriums and  2 ventricles..   it is possible for only one ventricle to hypertrophy... but this always leads to backed up problems of pressure... IE/  increased hydrostatic pressure backed into the lung's capillaries.... leading to pulmonary edema.. aka another quick way to die      so to answer your question... yes the all other chambers can be of normal size w/ the left ventricle exclusively increasing in muscularity/size =bad bad bad    

Also due to the compromised conductivity of cardiac tissue from steroid use, the chances of recovery of normal heartbeat during myocardial infarctions is reduced also.
18  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 28, 2013, 02:55:09 PM
Induced growth of cardiomyocyctes is irreversible my friend.....as seen in cases of hypertrophy, where the ventricle is forced to increase contractility in order to provide more oxygenated blood due to higher demand....    Follow physics...power = length x tension ...   Something is going to have to give ...it was just a matter of time.. The human body can only compensate to a certain futile degree.....  All the abusive factors contributed to his accelerated demise...  And yes..numerous past bodybuilders of the 70s did indeed undergo bypass surgery of some form- although not the hypertrophic acceleration seen in generation GH coupled w other anabolic enhancements... 


Christ!! Someone who knows what they are talking about at last!!
19  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 28, 2013, 02:51:39 PM
There are many reasons for heart failure. Although the use of steroids might have been detrimental, they might have not been directly correlated to his death.

Lets look at the obvious facts. The man was overweight and carrying a large amount of mass, which makes it difficult for the heart.

Due to his over-eating and excess weight, he could have had:

.....a history of hypertension (elevated blood pressure) that lead to the heart having to work much harder to pump blood from the left ventricle out into his systemic circulation. This is one of the major causes for left-ventricular hypertrophy, whereby the left ventricle becomes thickened due to the added exertion needed to pump blood our of the heart. Over time, this causes the left ventricle to become weak and/or stiff which can lead to heart failure.

.....a history of elevated cholesterol that lead to atherosclerosis (clogging and hardening by way of fatty deposits) of the coronary arteries. Over time, with enough fatty buildup, you can end up with plaque that lines the arteries of the heart. If pieces of plaque break off, they can travel in the form of small clots, which can then interrupt the blow flow through the coronary arteries leading to a heart attack (myocardial infarct). The combination of these two things (high cholesterol & a history of heart attacks) can lead to a decline of the ventricles (left and right, but mostly left) pumping capacity which can result in heart failure.

These major issues aside, the guy could have had calcification or even prolapse of his heart valves, which can also over time lead to heart failure.

Drug use (illicit drugs), not necessarily steroid use, could also lead to hypertrophic cardiomyopathy. This is not to say that he was using any illicit drugs, but that is another potential.

Also, to corroborate what Royalty was saying, it is true (based on a limited amount of studies) that anabolic steroid use has been linked to weakened left ventricular function.

Studies to back this up, can be found here:

Serious cardiovascular side effects of large doses of anabolic steroids in weight lifters - http://eurheartj.oxfordjournals.org/content/17/10/1576.abstract

Long-Term Anabolic-Androgenic Steroid Use Is Associated With Left Ventricular Dysfunction - http://circheartfailure.ahajournals.org/content/3/4/472

Ultimately, what I think is important to realize is that no mere title, trophy, sponsorship, magazine cover or even various sets of spectating eyes are worth endangering your health in the long run. You only live once and what's important is to live life to your full potential.

May his family find peace in this time of turmoil..

"1"

Haha, classic denialist mentality stuff....

Steroids lead to permanent myocardial fibrosis and micro scarring leading to reduced efficiency, disturbed electrical conductivity and the leading cause of heart failure over any of the other issues that have have been highlighted above.
20  Getbig Main Boards / Gossip & Opinions / Re: Greg Kovacs dies at 44 on: November 28, 2013, 02:48:10 PM
Obese people also have enlarged hearts. I'm not dismissing the drugs, I understand that all the orals, GH and vet drugs hinder health, but I believe staying huge past 40 is what does it in for these guys.
The smart thing for these retired pros to do is stay on trt, maybe even a bit more than trt (wouldn't hurt much anyway) and eat less, stay lean, stay healthy.

FACT = Steroids irreversibly alter the structure of the heart increasing the chances of a fatal cardiac event.

Why bodybuilders cant accept this, instead of looking for other answers. I guess they cant accept the fact that they have fucked themselves up....
21  Getbig Main Boards / Gossip & Opinions / Re: Me doing heavy front squats on: November 22, 2013, 04:00:12 PM
Good evening ladies and gentlemen

<a href="http://www.youtube.com/watch?v=T7rY9IC2HY8" target="_blank">http://www.youtube.com/watch?v=T7rY9IC2HY8</a>

I've been bulking recently, gained a little bit of fat:



What do you think, getbig?


lifting a heavy weight up and down, how fascinating, keeps some people interested for decades.....
22  Getbig Main Boards / Gossip & Opinions / Re: My Transformation on: November 22, 2013, 03:58:02 PM
What do you think he is on?

Nothing, if he has been training for a few years during his teens years, this is easily achievable. He will have recruited additional satellite cells beyond what an untrained teenager would have and will retain this development for life, or at least the muscle memory of it.
23  Getbig Main Boards / Gossip & Opinions / Re: Jameis Winston... on: November 22, 2013, 03:52:19 PM


 Grin Bodybuilding gold right there.

All thats missing is some fire breathing dragons and a heavy metal band.
24  Getbig Main Boards / Gossip & Opinions / Re: Getbig Memes on: November 22, 2013, 03:48:56 PM
Sign the Waiver
Option D
Dubai, India
Are there things?
Hebrew
tiny-tit (invented by getbig?)
Huh

Huh

Clambutteroil
25  Getbig Main Boards / Gossip & Opinions / Re: My Transformation on: November 22, 2013, 03:47:22 PM


excellent reduction in telomere length.
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