blood glucose drops because glucose is stuffed into muscle in the form of glycogen. insulin makes you fat, only when you are eating fat.
lol dude i dont care how many posts you have or what guys pic is on your avatar - you are a FOOL. i truely hope that no 'young buck' on this board listens to anything you say.
taken from:
http://www.vivo.colostate.edu/hbooks/pathphys/endocrine/pancreas/insulin_phys.htmlInsulin and Lipid Metabolism
The metabolic pathways for utilization of fats and carbohydrates are deeply and intricately intertwined. Considering insulin's profound effects on carbohydrate metabolism, it stands to reason that insulin also has important effects on lipid metabolism. Important effects of insulin on lipid metabolism include the following:
Insulin promotes synthesis of fatty acids in the liver. As discussed above, insulin is stimulatory to synthesis of glycogen in the liver. However, as glycogen accumulates to high levels (roughly 5% of liver mass), further synthesis is strongly suppressed.
When the liver is saturated with glycogen, any additional glucose taken up by hepatocytes is shunted into pathways leading to synthesis of fatty acids, which are exported from the liver as lipoproteins. The lipoproteins are ripped apart in the circulation, providing free fatty acids for use in other tissues, including adipocytes, which use them to synthesize triglyceride.
Insulin inhibits breakdown of fat in adipose tissue by inhibiting the intracellular lipase that hydrolyzes triglycerides to release fatty acids.
Insulin facilitates entry of glucose into adipocytes, and within those cells, glucose can be used to synthesize glycerol. This glycerol, along with the fatty acids delivered from the liver, are used to synthesize triglyceride within the adipocyte. By these mechanisms, insulin is involved in further accumulation of triglyceride in fat cells.
From a whole body perspective, insulin has a fat-sparing effect. Not only does it drive most cells to preferentially oxidize carbohydrates instead of fatty acids for energy, insulin indirectly stimulates accumulation of fat in adipose tissue.
taken from:
http://weightoftheevidence.blogspot.com/2005/09/insulin-fat-storage-fat-use-for-energy.htmlInsulin: Fat Storage - Fat Use for Energy
Researchers at the University of California, San Diego (UCSD) School of Medicine have reported in RxPG News that chronically high levels of insulin, as is found in many people with obesity and Type II diabetes, may block specific hormones that trigger energy release into the body. In other words, high insulin levels inhibit the use of body fat for energy in the body.
The researchers found in their studies that high levels of insulin can block stress hormones known as catecholamines, which normally cause the release of cellular energy. Adrenaline is the best known example of a catecholamine. For normal metabolism to occur, the body needs a balanced input of insulin and catecholamines. One of the actions of insulin --, the main energy storage hormone, is to block activation of the protein kinase A (PKA) enzyme. After a meal, insulin levels go up, and the body stores energy primarily as triglycerides, or fat, in adipose tissue to be used later. When energy is needed, catecholamine triggers activation of PKA, and energy is released. But in people with Type II diabetes, the hormonal balance has been thrown off, because the body continues to produce and store more triglyceride instead of breaking down the fat as released energy.
The findings provide additional understanding to the cause and effect occurring when insulin levels are chronically too high. We know that as insulin levels go up and the body loses the ability to effectively use it, so it makes more, bringing insulin levels even higher as the body struggles with what is called insulin resistence. Insulin resistence is a pre-cursor to Type II diabetes.
Overweight and obesity is seen in the vast majority of those with insulin resistence and Type II diabetes due to the chronic storage of fat in the body.
“If insulin levels get too high for too long a time – which happens in many patients with type II diabetes –the normal catecholamine signal that triggers fat breakdown and energy release can be drowned out. This can lead to excessive energy storage in the adipocyte,” said Hupfeld, assistant professor of Medicine in the UCSD Division of Endocrinology and Metabolism and a co-author of the paper. “This may be one reason why chronic obesity and Type II diabetes are often seen together.”
In lay terms, one gets fatter as their ability to effectively use insulin diminishes and their body makes more insulin, thus storing more energy as fat, in an effort to compensate for the insulin resistence. It really is a vicious cycle.
taken from:
http://www.musculardevelopment.com/content/view/510/54/Everyone knows that insulin has potent anabolic effects in the body. One thing that is greatly misconstrued is its method of action in this respect.
Insulin is not essentially anabolic to muscle tissue. However, insulin is anabolic to adipose tissue (fat). But, with regard to protein, insulin's anabolic action comes from its potent anti-catabolic effect. That is, protein breakdown is greatly reduced in the presence of insulin, thus accounting for insulin's anti-catabolic actions.
That is, insulin increases the production of ACC2, causing a blunted effect in fatty acids being transferred to the mitochondria. This results in less fat being burned for energy. When insulin is elevated in the body, fat loss is blunted because of increased ACC2.
Insulin may thwart low-fat diet:
http://www.cnn.com/2008/HEALTH/dailydose/11/20/insulin.diet/index.htmlhttp://health.howstuffworks.com/fat-cell2.htmHigh Insulin Levels Stop Fat Loss and Cause Weight Gain:
http://fitnessblackbook.com/dieting_for_fat_loss/high-insulin-levels-stop-fat-loss-and-cause-weight-gain/there are hundred more but i am sick of copying pasting.
tboner you are just wrong, and NO amount of arguing, opinions, or feelings is going to change human biology.
