He says that clen & ephedrine work by suppressing appetite. Really? 
Clen does this?
Yes. Really. Beta-2 stimulation is well known to slow gastric emptying, and activation of the sympathetic nervous system decreases hunger. Again, this is so well known and documented that it doesn't merit debate.
He says that it's unfortunate that clen decreases insulin sensitivity. Does he even know how clen works and why the insulin resistance is desirable? 
Why is insulin resistance desirable? You've peaked my interest. Insulin resistance is linked with heart problems as well as obesity. Tell me why that's desirable. Diabetics are insulin resistant. Is being diabetic desirable? Elaborate.
Next he talks about clen as a repartitioning agent. Okay, he says it may increase FFM, but the study that he references is on "chronic administration of therapeutic levels of clenbuterol acts as a repartitioning agent"... "in twenty-three unfit Standardbred mares!" (that’s adult female horses in case you didn’t know 
).
In the case of clenbuterol, animal data is basically all we have to go on.
Then he goes on to give us the graphs of the horse data. Apparently he hasn’t heard clen doesn't have {significant} anabolic effects in humans. It’s scary that he is using horse data to recommend full 6-12 weeks on. (because the mares who exercised only gained FFM (a whopping 4.4%) after week 6. You've got to be kidding.
If that study or my comments on it are invalid, please give a better reason than "you've got to be kidding me".
In other words, "even though I know it doesn't work like this in humans, I'm sure it works the same." WTF 
No. That's not what is says at all. Your creating a strawman, and an informal fallacy.
Now the ‘expert’ advice. "if you want the quasi-anabolic effect from the clen, it’ll take more than 2 weeks on (apparently 6 weeks)." Great advice. I'm sure it'll be much more anabolic at week 6 when your receptors are severely down regulated. He goes on, "Keep in mind that animal responses to beta-agonist/antagonists differ a bit from ours…(no f'g $h!t) but I’m sure that you get the idea that 2on/2off is not a great dosing protocol." Why do you even say to keep it in mind when recommending dosages based on a horses response?
Again, animal data is what we have to go on, with regards to references in medical journals. Oh...and the literally hundreds of members of steroid.com who have tried things my way and found it to be optimal and anabolic. And my dosing protocol is based on anecdotal human data (the members I surveyed and polled on steroid.com)...not just animal data. You should at least learn what kind of data I have/had access to before you attempt to dispute my conclusions.
He says, "The reasons for the further increase in FFM around week 6 despite no changes in % fat or fat mass is not easily explained." No? Well… let me explain it for you. No change in % fat BECAUSE THE RECEPTORS ARE FRIED!
Hence my reccomendations to include benadryl.
He says that clen will increase muscle protein synthesis in your body. Oh really? This is based off of the study he references - "Chronic effects of beta 2-adrenergic agonists on body composition and protein synthesis in the rat."
Ha ha. Again, beta-2 agonists are well known to have this effect. Your lack of understanding and research is showing.
Next says that "clenbuterol can help blast you past 'sticking points' in your training by circumventing the usual mechanisms by which anabolism is experienced! It is of note that both muscle composition and fiber size has been shown to increase with administration of clen." That study: "The effect of the anabolic agent, clenbuterol, on overloaded rat skeletal muscle."
See comment above.
Finally he says something about humans - "This type of dramatic anabolic effect hasn’t been confirmed in human studies." DUH!
Now you criticize me for agreeing with you. At least you don't need to be consistent in your terrible rambling asessment of my work.
You can read the rest if you want. For me, this confirms what I said earlier. The article is full of CRAP. This leads me to another observation (albeit speculative) – this guy probably shouldn’t be trusted since he uses random, inapplicable studies frequently.
I use the best data available within the limitations of what the medical field has to offer, combined with extensive polls and surveys on steroid.com, added to literally hundreds of data from athletes I interact with.
OK, I’ll shut up now.
Please don't. I'd like to see a valid refutation of my article, and not just "give me a break" and "duh" offered as your critique. If my references are invalid, please prove them so by counterexample or other studies. If you've ever researched clen (which you haven't), you'd know that human data is very scarse, and animal data is all we have to go on. In light of this, I suppose a detailed theory of why animal reaction to beta-2 stimulation would differ from the human reaction to it would be appropriate. I'll wait for your reply. But honestly, since your post shows an almost complete lack of understanding of the beta-2 system, I doubt anything you come up with will be sound...like the rest of your post.
Most interestingly, I'd like to learn how insulin resistance is desirable. Cardiovascular problems, increased risk of cancer, diabetes, shorter life spans, and obesity is desirable? That's the first thing I'd like explained, because I think that it's the thing that most accurately displays your total lack of understanding of...anything.
