Before you attempt to type out a mediocre response filled with caps and zeros in place of the letter "O" check your facts moron..
You are DEAD wrong.
Here are a couple of articles from refutable sources:
Straight from Harvard:
"Too much protein may cause reduced kidney function"
http://news.harvard.edu/gazette/2003/03.13/09-kidney.html
Cleveland Clinic:
http://www.atkinsexposed.org/atkins/114/cleveland_clinic.htm
From the Mayo Clinic:
"A high-protein diet may cause or worsen liver or kidney problems "
http://www.mayoclinic.com/health/high-protein-diets/AN00847
http://www.bmj.com/content/325/7361/408.5.extract
Effect of high protein diet on stone-forming propensity and bone loss in rats.Amanzadeh J, Gitomer WL, Zerwekh JE, Preisig PA, Moe OW, Pak CY, Levi M.
Center for Mineral Metabolism and Clinical Research and Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas 75390-8885, USA.
Comment in:
Kidney Int. 2004 May;65(5):1970; author reply 1970.
Abstract
BACKGROUND: High protein diets are believed to cause kidney stone formation and bone loss, but the mechanisms mediating these changes are unknown. The purpose of this study was to create an animal model of animal protein excess and to evaluate the response of kidney and bone to the dietary protein load.
METHODS: Rats (12 per group) were pair-fed with a high (48%) and low (12%) casein diets that were otherwise identical in their content of sodium, potassium, calcium, phosphorus, and magnesium.
RESULTS: Compared with the low casein group, the high casein group delivered a substantial acid load during 59 days of study, since it significantly decreased urinary pH, and increased urinary ammonium, titratable acidity, and net acid excretion. Animals on high casein diet also had higher urinary volumes. On the high casein diet, urinary calcium excretion was significantly higher and urinary citrate excretion and concentration was significantly lower. On the high casein diet, urinary saturation of calcium phosphate was higher. Serum calcitriol concentration did not significantly differ between the two groups. Histomorphometric analysis of femur procured after 59 days on the diet showed marked increase in bone resorption in the high casein group. Hypocitraturia was associated with increased activity of sodium-citrate cotransporter in renal cortical brush-border membranes (BBM) in the high casein group.
CONCLUSION: Both the kidney and bone contribute to the pathogenesis of hypercalciuria during high casein diet in rats. Hypocitraturia is probably renal in origin. This rat model will be useful in elucidating the mechanisms by which high protein intake increases the risk of nephrolithiasis and bone loss in human beings.
This piece: "Kidney failure. Consuming too much protein puts a strain on the kidneys, which can make a person susceptible to kidney disease."
~ obtained from: http://www.atkinsexposed.org/atkins/114/cleveland_clinic.htm
Another refutable source: Brenner, B. Dietary protein intake and the progressive nature of kidney disease. N. Engl. J. Med. 307:652, 1982
Now shut the fuck up and go crawl under that rock you came out of, I love how these little pussies always like to talk without any backing of their claims.
"1"
Y0U ARE A FUCKING M0R0N AND D0 N0T UNDERSTAND WHAT MEDICAL LITERATURE IS.
Y0U FIRST STUDY LET'S SEE. Researchers at Brigham and Women’s Hospital (BWH) have found that high-protein diets may be associated with kidney function decline in women who already have mildly reduced kidney function. IN MIDLY REDUCED KIDNEY FUNCTI0N. D0 Y0U UNDERSTAND WHAT THIS MEANS? THIS MEANS THEIR KIDNEYS ARE ALREADY DAMAGED. I'M TALKING AB0UT HEALTHY INDIVIDUALS. Y0U SEC0ND ARTICLE. DR GRAYS0N CLEVELAND CLINIC GIVING HIS FALSE 0PINI0N WITH N0 STUDY D0NE? ARE Y0U SERI0US. GTF0 0UT HERE WITH THAT 0NE. Y0UR LAST 0NE. DR GRAYS0N SH0ULD N0T EVEN BE A D0CT0R BECAUSE HE HAS N0 IDEA WHAT KET0SIS IS.
L0L AT KET0SIS BEING DANGER0US IN HEALTHY INDIVIDUALS. KET0SIS IS N0T DANGER0US AT ALL AND THE BRAINS L0VES KET0NES 0NCE ADAPTED T0 THEM. LET'S SEE WHAT R0BERT VEECH AN ACTUAL MEDICAL D0CT0R HAD T0 SAY AB0UT THIS. VEECH IS THE W0RLD'S LEADING RESEARCHER 0N KET0NES.
I quote the article: " 'Doctors are scared of ketosis,' says Richard Veech, an N.I.H. [National Institutes of
Health]researcher who studied medicine at Harvard and then got his doctorate at Oxford University with
the Nobel Laureate Hans Krebs. ''They're always worried about diabetic ketoacidosis. But ketosis is a normal
physiologic state."
"Simply put, ketosis is evolution's answer to the thrifty gene. We may have evolved to efficiently store fat for
times of famine, says Veech, but we also evolved ketosis to efficiently live off that fat when necessary. Rather
than being poison, which is how the press often refers to ketones, they make the body run more efficiently
and provide a backup fuel source for the brain. Veech calls ketones ''magic'' and has shown that both the
heart and brain run 25 percent more efficiently on ketones than on blood sugar."
http://www.medicalvitadiet.com.au/docs/vita_ketosis.pdfHigh protein diet brings risk of kidney stones
Scott Gottlieb
+ Author Affiliations
New York
Diets heavy on foods that are high in protein and low in carbohydrates can increase the risk of kidney stones and reduce the body's ability to absorb calcium after just six weeks.
INCREASE THE RISK FR0M KIDNEY ST0NES AFTER 6 WEEKS? L0L. IF THAT'S THE CASE THEN EVERY0NE HERE AND EVERY B0DYBUILDER SH0ULD HAVE 0NE KIDNEY REMAINING. IN DAMAGED KIDNEYS, YES IT CAN. IN HEALTHY INDIVIDUALS? N0 IT CAN'T.
These findings from a new study come at a time when an increasing number of Americans, seduced by anecdotal accounts of fast weight loss, are turning to low carbohydrate, high protein diets. Popularised by Dr Robert Atkins, these diets are having a revival after a recent cover story in the New … CAN'T EVEN SEE THE FULL TEXT. I'M BETTING THEY ARE D0NE 0N PE0PLE WITH REDUCED KIDNEY FUNCTI0N AGAIN. I'M STILL WAITING F0R Y0U T0 SH0W ME A STUDY D0NE 0N HEALTHY INDIVIDUALS AND N0T PE0PLE WITH REDUCED KIDNEY FUNCTI0NS. Y0U DUMB FUCK. TYPICAL 0F UNEDUCATED PE0PLE P0STING AN 0PINI0N 0F A D0CT0R WITH N0 STUDY T0 BACK IT UP FR0M THE CLEVELAND CLINIC. IN FACT EVERY STUDY THAT WAS D0NE 0N A HIGH PR0TEIN HIGH FAT L0W CARB DIET IMPR0VED BL00D LIPIDS AND DECREASED B0DY FAT. HIGH GLUC0SE LEVELS 0N THE 0THER HAND FR0M EXCESSIVE INTAKE 0F SUGAR AND CARBS HAVE BEEN SH0WN T0 DAMAGE THE KIDNEYS.
REGARDING THE RATS. THAT IS JUST T00 FUNNY. AN INCREASE IN ACID LEVELS. W0RKING 0UT CAUSE THE M0ST AM0UNT 0F ACID IN Y0UR B0DY. THAT STUDY DID N0T PR0VE ANYTHING AB0UT CERTAIN PR0TEIN LEVELS DAMAGING HUMAN KIDNEYS IN HEALTHY INDIVIDUALS. TRY AGAIN. VERY P00R STUDY 0N THE RATS. HERE LET ME SH0W Y0U 0NE.
http://ajpregu.physiology.org/content/287/4/R934.shortA long-term high-protein diet markedly reduces adipose tissue without major side effects in Wistar male rats
Magali Lacroix,1 Claire Gaudichon,1 Antoine Martin,2 Céline Morens,1 Véronique Mathé,1 Daniel Tomé,1 and Jean-François Huneau1
1Institut National de la Recherche Agronomique, Unité de Physiologie de la Nutrition et du Comportement alimentaire, Institut National Agronomique Paris-Grignon, 75231 Paris cedex 05; and 2Service d'Anatomie Pathologique, Hôpital Avicenne, 93000 Bobigny, France
Submitted 13 February 2004 ; accepted in final form 14 May 2004
Although there is a considerable interest of high-protein, low-carbohydrate diets to manage weight control, their safety is still the subject of considerable debate. They are suspected to be detrimental to the renal and hepatic functions, calcium balance, and insulin sensitivity. However, the long-term effects of a high-protein diet on a broad range of parameters have not been investigated. We studied the effects of a high-protein diet in rats over a period of 6 mo. Forty-eight Wistar male rats received either a normal-protein (NP: 14% protein) or high-protein (HP: 50% protein) diet. Detailed body composition, plasma hormones and nutrients, liver and kidney histopathology, hepatic markers of oxidative stress and detoxification, and the calcium balance were investigated. No major alterations of the liver and kidneys were found in HP rats, whereas NP rats exhibited massive hepatic steatosis. The calcium balance was unchanged, and detoxification markers (GSH and GST) were enhanced moderately in the HP group. In contrast, HP rats showed a sharp reduction in white adipose tissue and lower basal concentrations of triglycerides, glucose, leptin, and insulin. Our study suggests that the long-term consumption of an HP diet in male rats has no deleterious effects and could prevent metabolic syndrome.
hormones; glucose; body composition; histology
THEY ATE A DIET 0F 50 PERCENT PR0TEIN WITH N0 SIDE EFFECTS F0R 6 FUCKING M0NTHS. 50 FUCKING PERCENT. D0 Y0U KN0W H0W MUCH PR0TEIN THAT IS? EVERYTHING DECREASED FR0M BL00D LEVELS T0 GLUC0SE LEVELS WITH N0 KIDNEY DAMAGE.THIS IS AN ACTUAL STUDY THAT MEASURED L0NG TERM RESULTS AND DIDN'T TALK AB0UT RISKS WHICH ARE ALL BASELESS. Y0U ARE JUST A PLAIN ZEAL0T THAT'S ALL. D0N'T TRY T0 DEBATE WITH ME 0N THIS T0PIC 0R ANYTHING AB0UT NUTRITI0N. I KN0W M0RE AB0UT THIS THEN M0ST PE0PLE.