Author Topic: The Frutarian - eat nothing but fruit for 6 years  (Read 28435 times)

suckmymuscle

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #25 on: November 27, 2011, 02:27:25 AM »
  People who claim that fructose causes artherosclerosis and insulin resistence have no idea how carbohydrate metabolism works. They extrapolate the results of extremely high doses of fructose given to mice, who are omniverous rodents with poor carbohydrate processing ability, to humans and think that eating apples will give you diabetes and artherosclerosis. Subjects on high fructose diets who show fat deposition in the liver and diabetes are no different from any other people who eat tons of calories from protein, fat or other monosaccharides and polysaccharides. In fact fructose increases carbohydrate processing efficiency by signaling the activation of glucokinase. To prove this, besides the two studies I posted that show no link between fructose and metabolic syndrome in humans, here is an explanation about how monosaccharide metabolism works in humans. Unlike 99.9% of everything that people write about nutrition, this is from a hardcore physiology textbook. Scientific fact and not fiction:

  http://www.medbio.info/Horn/Time%201-2/carbohydrate_metabolism.htm

SUCKMYMUSCLE

suckmymuscle

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #26 on: November 27, 2011, 02:36:38 AM »
  The two double-blind, placebo-controlled studies showing no relation between fructose consumption and increased plasma triglycerides and hyperglycemia on humans, assumptions that were made incorrectly from measuring the plasma triglyceride and glucose levels of mice given extremely high levels of fructose:

  http://www.informaworld.com/smpp/jump~jumptype=banner~frompagename=content~frommainurifile=content~fromdb=all~fromtitle=~fromvnxs=~cons=?dropin=dxdoiorg_101080_104083982010512990&to_url=http%3a%2f%2fdx%2edoi%2eorg%2f10%2e1080%2f10408398%2e2010%2e512990

  http://www.informaworld.com/smpp/jump~jumptype=banner~frompagename=content~frommainurifile=content~fromdb=all~fromtitle=~fromvnxs=~cons=?dropin=dxdoiorg_101080_10408390903461426&to_url=http%3a%2f%2fdx%2edoi%2eorg%2f10%2e1080%2f10408390903461426

SUCKMYMUSCLE

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #27 on: November 27, 2011, 02:57:54 AM »
Thanks for posting this thread, this dude is cool, I have watched some more of his vids on his youtube channel - Great attitude
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Re: The Frutarian.
« Reply #28 on: November 27, 2011, 12:22:12 PM »
 All the studies that showed increased plasma levels of triglycerides from fructose were seen on those consuming massive amounts of high-fructose corn syrup in the form of soda and processed goods. Also, it has never been conclusively shown on humans. Most of the studies were done on rhesus monkeys and rats, which are metabolically a lot less efficient in processing fructose than humans. For whatever reasons, the human metabolism is extremely efficient in processing monosaccharides.

http://www.informaworld.com/smpp/jump~jumptype=banner~frompagename=content~frommainurifile=content~fromdb=all~fromtitle=~fromvnxs=~cons=?dropin=dxdoiorg_101080_104083982010512990&to_url=http%3a%2f%2fdx%2edoi%2eorg%2f10%2e1080%2f10408398%2e2010%2e512990

  http://www.informaworld.com/smpp/jump~jumptype=banner~frompagename=content~frommainurifile=content~fromdb=all~fromtitle=~fromvnxs=~cons=?dropin=dxdoiorg_101080_10408390903461426&to_url=http%3a%2f%2fdx%2edoi%2eorg%2f10%2e1080%2f10408390903461426

SUCKMYMUSCLE

high fructose corn syrup is fructose, it is simply fructose mixed with another sugar mostly glucose, so the data still apply. I like this quote from one of the papers showing negative effects on renal health "We suggest excessive fructose intake should be considered an environmental toxin with major health implications". so we know fructose impairs leptin sensitivity, increases obesity, can induce non-alcoholic fatty liver disease, increases LDL, impairs renal function and promotes insulin resistence. It is clearly not a healthy sugar to consume any large quantities of, i believe that is clear, that is the general consensus of the scientific community.


3 Address reprint requests to PJ Havel, Department of Nutrition, University of California, Davis, One Shields Avenue, Davis, CA 95616. E-mail: pjhavel@ucdavis.edu.


ABSTRACT
This review explores whether fructose consumption might be a contributing factor to the development of obesity and the accompanying metabolic abnormalities observed in the insulin resistance syndrome. The per capita disappearance data for fructose from the combined consumption of sucrose and high-fructose corn syrup have increased by 26%, from 64 g/d in 1970 to 81 g/d in 1997. Both plasma insulin and leptin act in the central nervous system in the long-term regulation of energy homeostasis. Because fructose does not stimulate insulin secretion from pancreatic ß cells, the consumption of foods and beverages containing fructose produces smaller postprandial insulin excursions than does consumption of glucose-containing carbohydrate. Because leptin production is regulated by insulin responses to meals, fructose consumption also reduces circulating leptin concentrations. The combined effects of lowered circulating leptin and insulin in individuals who consume diets that are high in dietary fructose could therefore increase the likelihood of weight gain and its associated metabolic sequelae. In addition, fructose, compared with glucose, is preferentially metabolized to lipid in the liver. Fructose consumption induces insulin resistance, impaired glucose tolerance, hyperinsulinemia, hypertriacylglycerolemia, and hypertension in animal models. The data in humans are less clear. Although there are existing data on the metabolic and endocrine effects of dietary fructose that suggest that increased consumption of fructose may be detrimental in terms of body weight and adiposity and the metabolic indexes associated with the insulin resistance syndrome, much more research is needed to fully understand the metabolic effect of dietary fructose in humans.

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC552336/?tool=pmcentrez

this paper shows elevated LDL cholesterol and triglycerides.


Nat Clin Pract Endocrinol Metab. 2006 Aug;2(8):447-58.
Childhood obesity: behavioral aberration or biochemical drive? Reinterpreting the First Law of Thermodynamics.
Lustig RH.
Source
Division of Endocrinology, University of California San Francisco, San Francisco, CA 94143-0434, USA. rlustig@peds.ucsf.edu

this shows its negative effect on leptin.

http://www.ncbi.nlm.nih.gov/pubmed/16076983

rat study on obesity

^ Hughes TA, Atchison J, Hazelrig JB, Boshell BR (1989). "Glycemic responses in insulin-dependent diabetic patients: effect of food composition". Am. J. Clin. Nutr. 49 (4): 658–66. PMID 2929488.
^ Wylie-Rosett, Judith; et al. (2004). "Carbohydrates and Increases in Obesity: Does the Type of Carbohydrate Make a Difference?". Obesity Res 12: 124S–129S. doi:10.1038/oby.2004.277. PMID 15601960.
^ a b Havel PJ (2001). "Peripheral signals conveying metabolic information to the brain: short-term and long-term regulation of food intake and energy homeostasis". Exp. Biol. Med. (Maywood) 226 (11): 963–77. PMID 11743131.
^ Dennison BA, Rockwell HL, Baker SL (1997). "Excess fruit juice consumption by preschool-aged children is associated with short stature and obesity". Pediatrics 99 (1): 15–22. PMID 8989331.

studies in humans showing the negative effects of fructose on insulin resistence and obesity, in humans, with fructose only.


another human study with just fructose, it's not looking good.

Am J Clin Nutr. 2000 Nov;72(5):1128-34.
Effects of dietary fructose on plasma lipids in healthy subjects.
Bantle JP, Raatz SK, Thomas W, Georgopoulos A.
Source
Department of Medicine, the General Clinical Research Center, the Division of Biostatistics, and the School of Public Health, the University of Minnesota, Minneapolis, MN 55455, USA. bantl001@tc.umn.edu
Abstract
BACKGROUND:
About 9% of average dietary energy intake in the United States comes from fructose. Such a high consumption raises concern about the metabolic effects of this sugar.

OBJECTIVE:
The objective of this study was to determine the effect of dietary fructose on plasma lipids.

DESIGN:
The study was conducted in the General Clinical Research Center at Fairview-University of Minnesota Medical Center. The participants were 24 healthy adult volunteers (12 men and 12 women; 6 of each sex were aged <40 y and 6 of each sex were aged >/=40 y). All subjects received 2 isoenergetic study diets assigned by using a randomized, balanced crossover design. One diet provided 17% of energy as fructose. The other diet was sweetened with glucose and was nearly devoid of fructose. Each diet was fed for 6 wk. Both diets were composed of common foods and contained nearly identical amounts of carbohydrate, protein, fat, fiber, cholesterol, and saturated, monounsaturated, and polyunsaturated fatty acids. All meals were prepared in the metabolic kitchen of the General Clinical Research Center.

RESULTS:
The responses to the study diets differed by sex. In men, the fructose diet produced significantly higher fasting, postprandial, and daylong plasma triacylglycerol concentrations than did the glucose diet. The daylong plasma triacylglycerol concentration after 6 wk of the fructose diet was 32% greater in men than the corresponding concentration during the glucose diet (P: < 0.001). The fructose diet had no significant effect on fasting or postprandial plasma triacylglycerol concentrations in women. The fructose diet also had no persistent effect on fasting plasma cholesterol, HDL cholesterol, or LDL cholesterol in either men or women.

CONCLUSIONS:
Dietary fructose was associated with increased fasting and postprandial plasma triacylglycerol concentrations in men. Diets high in added fructose may be undesirable, particularly for men. Glucose may be a suitable replacement sugar.

another human trial, liver issues


http://www.enerex.ca/en/articles/whey-protein-and-fructose-an-unhealthy-combination

http://www.westonaprice.org/modern-foods/murky-world-of-hfcs


It inducinG NAFLD

BACKGROUND/AIMS
While the rise in non-alcoholic fatty liver disease (NAFLD) parallels the increase in obesity and diabetes, a significant increase in dietary fructose consumption in industrialized countries has also occurred. The increased consumption of high fructose corn syrup, primarily in the form of soft-drinks, is linked with complications of the insulin resistance syndrome. Furthermore, the hepatic metabolism of fructose favors de novo lipogenesis and ATP depletion. We hypothesize that increased fructose consumption contributes to the development of NAFLD.
METHODS
A dietary history and paired serum and liver tissue were obtained from patients with evidence of biopsy-proven NAFLD (n=49) without cirrhosis and controls (n=24) matched for gender, age (± 5 years), and body mass index (± 3 points).
RESULTS
Consumption of fructose in patients with NAFLD was nearly 2-3 fold higher than controls [365 kcal. vs 170 kcal (p<0.05)]. In patients with NAFLD (n=6), hepatic mRNA expression of fructokinase (KHK), an important enzyme for fructose metabolism, and fatty acid synthase, an important enzyme for lipogenesis were increased (p=0.04 and p=0.02 respectively). In an AML hepatocyte cell line, fructose resulted in dose-dependent increase in KHK protein and activity.
CONCLUSION
The pathogenic mechanism underlying the development of NAFLD may be associated with excessive dietary fructose consumption.

here again showing avoidance of fructose poses many benefits

http://www.ncbi.nlm.nih.gov/pubmed/21523663

Lipids Health Dis. 2011 Jan 24;10:20.
Fructose impairs glucose-induced hepatic triglyceride synthesis.
Huang D, Dhawan T, Young S, Yong WH, Boros LG, Heaney AP.
Source
Department of Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA.
Abstract
Obesity, type 2 diabetes and hyperlipidemia frequently coexist and are associated with significantly increased morbidity and mortality. Consumption of refined carbohydrate and particularly fructose has increased significantly in recent years and has paralled the increased incidence of obesity and diabetes. Human and animal studies have demonstrated that high dietary fructose intake positively correlates with increased dyslipidemia, insulin resistance, and hypertension. Metabolism of fructose occurs primarily in the liver and high fructose flux leads to enhanced hepatic triglyceride accumulation (hepatic steatosis). This results in impaired glucose and lipid metabolism and increased proinflammatory cytokine expression. Here we demonstrate that fructose alters glucose-stimulated expression of activated acetyl CoA carboxylase (ACC), pSer hormone sensitive lipase (pSerHSL) and adipose triglyceride lipase (ATGL) in hepatic HepG2 or primary hepatic cell cultures in vitro. This was associated with increased de novo triglyceride synthesis in vitro and hepatic steatosis in vivo in fructose- versus glucose-fed and standard-diet fed mice. These studies provide novel insight into the mechanisms involved in fructose-mediated hepatic hypertriglyceridemia and identify fructose-uptake as a new potential therapeutic target for lipid-associated diseases

another

J Am Soc Nephrol. 2010 Dec;21(12):2036-9. Epub 2010 Nov 29.
The effect of fructose on renal biology and disease.
Johnson RJ, Sanchez-Lozada LG, Nakagawa T.
Source
Division of Renal Diseases and Hypertension, University of Colorado, Aurora, Colorado 80045, USA. richard.johnson@ucdenver.edu
Abstract
Dietary fructose intake is increasing. It is increasing primarily from added sugars, including sucrose and high fructose corn syrup, and correlates epidemiologically with the rising prevalence of metabolic syndrome and hypertension worldwide. The administration of fructose to animals and humans increases BP and the development of metabolic syndrome. These changes occur independently of caloric intake because of the effect of fructose on ATP depletion and uric acid generation. Fructose ingestion may also be a risk factor for kidney disease that includes glomerular hypertension, renal inflammation, and tubulointerstitial injury in animals. We suggest excessive fructose intake should be considered an environmental toxin with major health implications


Dr Dutch

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Re: The Frutarian.
« Reply #29 on: November 27, 2011, 12:23:50 PM »
Been eating 6-8 servings of raw fruit and veggies for the last 15 years. That's why I look decades younger than my peers pears.
fixed

dustin

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Re: The Frutarian.
« Reply #30 on: November 27, 2011, 12:25:41 PM »
I'm not sure he gets his daily 1.5 grams of protein per lbs of bodyweight.... :D

Don't worry, he probably gets it straight from the tap.

Necrosis

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Re: The Frutarian.
« Reply #31 on: November 27, 2011, 12:25:48 PM »

so fruit is poison lol idiot

no, didnt say that but not that i expect you to have critical thinking skills. Fructose which is one of the sugars in fruits (varying degrees) is not a healthy sugar. There are things in fruits that are not healthy and not healthy for certain individuals. several servings of fruits a day is healthy due to the vitamins, phytochemicals etc. but having a fruit only or heavy diet is not, its just to much sugar which has all kinds of negative effects on health. Human physiology is such that meat and fatty acids are required in most cases. vegetables have more benefits then fruits while improving glucose regulation. Why do you think we have HCL in our stomachs, to digest carbs? lol.

aesthetics

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #32 on: November 27, 2011, 12:26:03 PM »
i dont care at all and he also looks like a homo and acts like one too.

devilsmile

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #33 on: November 27, 2011, 12:28:47 PM »
wow, by reading this thread I have learned that fruits are good for you, oh my goodness I feel so enlightenned  ::)

suckmymuscle

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Re: The Frutarian.
« Reply #34 on: November 27, 2011, 01:05:43 PM »
high fructose corn syrup is fructose, it is simply fructose mixed with another sugar mostly glucose, so the data still apply. I like this quote from one of the papers showing negative effects on renal health "We suggest excessive fructose intake should be considered an environmental toxin with major health implications". so we know fructose impairs leptin sensitivity, increases obesity, can induce non-alcoholic fatty liver disease, increases LDL, impairs renal function and promotes insulin resistence. It is clearly not a healthy sugar to consume any large quantities of, i believe that is clear, that is the general consensus of the scientific community.


3 Address reprint requests to PJ Havel, Department of Nutrition, University of California, Davis, One Shields Avenue, Davis, CA 95616. E-mail: pjhavel@ucdavis.edu.


ABSTRACT
This review explores whether fructose consumption might be a contributing factor to the development of obesity and the accompanying metabolic abnormalities observed in the insulin resistance syndrome. The per capita disappearance data for fructose from the combined consumption of sucrose and high-fructose corn syrup have increased by 26%, from 64 g/d in 1970 to 81 g/d in 1997. Both plasma insulin and leptin act in the central nervous system in the long-term regulation of energy homeostasis. Because fructose does not stimulate insulin secretion from pancreatic ß cells, the consumption of foods and beverages containing fructose produces smaller postprandial insulin excursions than does consumption of glucose-containing carbohydrate. Because leptin production is regulated by insulin responses to meals, fructose consumption also reduces circulating leptin concentrations. The combined effects of lowered circulating leptin and insulin in individuals who consume diets that are high in dietary fructose could therefore increase the likelihood of weight gain and its associated metabolic sequelae. In addition, fructose, compared with glucose, is preferentially metabolized to lipid in the liver. Fructose consumption induces insulin resistance, impaired glucose tolerance, hyperinsulinemia, hypertriacylglycerolemia, and hypertension in animal models. The data in humans are less clear. Although there are existing data on the metabolic and endocrine effects of dietary fructose that suggest that increased consumption of fructose may be detrimental in terms of body weight and adiposity and the metabolic indexes associated with the insulin resistance syndrome, much more research is needed to fully understand the metabolic effect of dietary fructose in humans.

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC552336/?tool=pmcentrez

this paper shows elevated LDL cholesterol and triglycerides.


Nat Clin Pract Endocrinol Metab. 2006 Aug;2(8):447-58.
Childhood obesity: behavioral aberration or biochemical drive? Reinterpreting the First Law of Thermodynamics.
Lustig RH.
Source
Division of Endocrinology, University of California San Francisco, San Francisco, CA 94143-0434, USA. rlustig@peds.ucsf.edu

this shows its negative effect on leptin.

http://www.ncbi.nlm.nih.gov/pubmed/16076983

rat study on obesity

^ Hughes TA, Atchison J, Hazelrig JB, Boshell BR (1989). "Glycemic responses in insulin-dependent diabetic patients: effect of food composition". Am. J. Clin. Nutr. 49 (4): 658–66. PMID 2929488.
^ Wylie-Rosett, Judith; et al. (2004). "Carbohydrates and Increases in Obesity: Does the Type of Carbohydrate Make a Difference?". Obesity Res 12: 124S–129S. doi:10.1038/oby.2004.277. PMID 15601960.
^ a b Havel PJ (2001). "Peripheral signals conveying metabolic information to the brain: short-term and long-term regulation of food intake and energy homeostasis". Exp. Biol. Med. (Maywood) 226 (11): 963–77. PMID 11743131.
^ Dennison BA, Rockwell HL, Baker SL (1997). "Excess fruit juice consumption by preschool-aged children is associated with short stature and obesity". Pediatrics 99 (1): 15–22. PMID 8989331.

studies in humans showing the negative effects of fructose on insulin resistence and obesity, in humans, with fructose only.


another human study with just fructose, it's not looking good.

Am J Clin Nutr. 2000 Nov;72(5):1128-34.
Effects of dietary fructose on plasma lipids in healthy subjects.
Bantle JP, Raatz SK, Thomas W, Georgopoulos A.
Source
Department of Medicine, the General Clinical Research Center, the Division of Biostatistics, and the School of Public Health, the University of Minnesota, Minneapolis, MN 55455, USA. bantl001@tc.umn.edu
Abstract
BACKGROUND:
About 9% of average dietary energy intake in the United States comes from fructose. Such a high consumption raises concern about the metabolic effects of this sugar.

OBJECTIVE:
The objective of this study was to determine the effect of dietary fructose on plasma lipids.

DESIGN:
The study was conducted in the General Clinical Research Center at Fairview-University of Minnesota Medical Center. The participants were 24 healthy adult volunteers (12 men and 12 women; 6 of each sex were aged <40 y and 6 of each sex were aged >/=40 y). All subjects received 2 isoenergetic study diets assigned by using a randomized, balanced crossover design. One diet provided 17% of energy as fructose. The other diet was sweetened with glucose and was nearly devoid of fructose. Each diet was fed for 6 wk. Both diets were composed of common foods and contained nearly identical amounts of carbohydrate, protein, fat, fiber, cholesterol, and saturated, monounsaturated, and polyunsaturated fatty acids. All meals were prepared in the metabolic kitchen of the General Clinical Research Center.

RESULTS:
The responses to the study diets differed by sex. In men, the fructose diet produced significantly higher fasting, postprandial, and daylong plasma triacylglycerol concentrations than did the glucose diet. The daylong plasma triacylglycerol concentration after 6 wk of the fructose diet was 32% greater in men than the corresponding concentration during the glucose diet (P: < 0.001). The fructose diet had no significant effect on fasting or postprandial plasma triacylglycerol concentrations in women. The fructose diet also had no persistent effect on fasting plasma cholesterol, HDL cholesterol, or LDL cholesterol in either men or women.

CONCLUSIONS:
Dietary fructose was associated with increased fasting and postprandial plasma triacylglycerol concentrations in men. Diets high in added fructose may be undesirable, particularly for men. Glucose may be a suitable replacement sugar.

another human trial, liver issues


http://www.enerex.ca/en/articles/whey-protein-and-fructose-an-unhealthy-combination

http://www.westonaprice.org/modern-foods/murky-world-of-hfcs


It inducinG NAFLD

BACKGROUND/AIMS
While the rise in non-alcoholic fatty liver disease (NAFLD) parallels the increase in obesity and diabetes, a significant increase in dietary fructose consumption in industrialized countries has also occurred. The increased consumption of high fructose corn syrup, primarily in the form of soft-drinks, is linked with complications of the insulin resistance syndrome. Furthermore, the hepatic metabolism of fructose favors de novo lipogenesis and ATP depletion. We hypothesize that increased fructose consumption contributes to the development of NAFLD.
METHODS
A dietary history and paired serum and liver tissue were obtained from patients with evidence of biopsy-proven NAFLD (n=49) without cirrhosis and controls (n=24) matched for gender, age (± 5 years), and body mass index (± 3 points).
RESULTS
Consumption of fructose in patients with NAFLD was nearly 2-3 fold higher than controls [365 kcal. vs 170 kcal (p<0.05)]. In patients with NAFLD (n=6), hepatic mRNA expression of fructokinase (KHK), an important enzyme for fructose metabolism, and fatty acid synthase, an important enzyme for lipogenesis were increased (p=0.04 and p=0.02 respectively). In an AML hepatocyte cell line, fructose resulted in dose-dependent increase in KHK protein and activity.
CONCLUSION
The pathogenic mechanism underlying the development of NAFLD may be associated with excessive dietary fructose consumption.

here again showing avoidance of fructose poses many benefits

http://www.ncbi.nlm.nih.gov/pubmed/21523663

Lipids Health Dis. 2011 Jan 24;10:20.
Fructose impairs glucose-induced hepatic triglyceride synthesis.
Huang D, Dhawan T, Young S, Yong WH, Boros LG, Heaney AP.
Source
Department of Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA 90095, USA.
Abstract
Obesity, type 2 diabetes and hyperlipidemia frequently coexist and are associated with significantly increased morbidity and mortality. Consumption of refined carbohydrate and particularly fructose has increased significantly in recent years and has paralled the increased incidence of obesity and diabetes. Human and animal studies have demonstrated that high dietary fructose intake positively correlates with increased dyslipidemia, insulin resistance, and hypertension. Metabolism of fructose occurs primarily in the liver and high fructose flux leads to enhanced hepatic triglyceride accumulation (hepatic steatosis). This results in impaired glucose and lipid metabolism and increased proinflammatory cytokine expression. Here we demonstrate that fructose alters glucose-stimulated expression of activated acetyl CoA carboxylase (ACC), pSer hormone sensitive lipase (pSerHSL) and adipose triglyceride lipase (ATGL) in hepatic HepG2 or primary hepatic cell cultures in vitro. This was associated with increased de novo triglyceride synthesis in vitro and hepatic steatosis in vivo in fructose- versus glucose-fed and standard-diet fed mice. These studies provide novel insight into the mechanisms involved in fructose-mediated hepatic hypertriglyceridemia and identify fructose-uptake as a new potential therapeutic target for lipid-associated diseases

another

J Am Soc Nephrol. 2010 Dec;21(12):2036-9. Epub 2010 Nov 29.
The effect of fructose on renal biology and disease.
Johnson RJ, Sanchez-Lozada LG, Nakagawa T.
Source
Division of Renal Diseases and Hypertension, University of Colorado, Aurora, Colorado 80045, USA. richard.johnson@ucdenver.edu
Abstract
Dietary fructose intake is increasing. It is increasing primarily from added sugars, including sucrose and high fructose corn syrup, and correlates epidemiologically with the rising prevalence of metabolic syndrome and hypertension worldwide. The administration of fructose to animals and humans increases BP and the development of metabolic syndrome. These changes occur independently of caloric intake because of the effect of fructose on ATP depletion and uric acid generation. Fructose ingestion may also be a risk factor for kidney disease that includes glomerular hypertension, renal inflammation, and tubulointerstitial injury in animals. We suggest excessive fructose intake should be considered an environmental toxin with major health implications



  I never denied that high-fructose corn syrup is fructose; my point being that the amounts given to the rats were enormous, and that rats, being omniverous scavengers, do not process sugars as well as we do since their diet is more composed of animal matter including decaying animal matter. You cannot extrapolate those studies to humans.

  As for the human studies you posted, the study I posted was done by several physiologists who analysed all the studies done and concluded that there is no conclusive evidence that fructose increases plasma triglycerides and insuling resistence in humans more than a diet with an equal amount of calories would. Funny that chimpanzees and lowland gorillas get 95% of their calories from fruit and yet never get any artherosclerosis or diabetes.

SUCKMYMUSCLE

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #35 on: November 27, 2011, 01:10:58 PM »
He has been eating nothing but fruit for 6 years. And yet, he has so much energy that he can run 4 marathons in a week, and his complexion glows with vitality. His blood work shows outstanding health: extremely low LDL cholesterol and high HDL cholesterol, extremely low levels of uric acid, amonia and creatinine in urine and the most amazing thing of all is that, even though he gets 90% of his calories from carbohydrates, he has extremely low levels of insulin. Fruit is rich in fructose, a monosaccharide that dramatically increases the body's ability to use glucose effectively. And yes, he is very skinny, but that is not due to the fruitarian diet but due to the fact that he runs 20 miles a day, every day. Meat-eating marathoners look exactly like him.

  I do not recommend this diet. Fruits are too low in EFAs and especially Calcium, Phosphorus, Iron and Zinc, and the little it does has very low bioavailability compared to the chelated minerals in animal proteins. But still, this diet is very good for the liver, kidneys, heart and vascular systems.

  


SUCKMYMUSCLE
  

sucky what are your thoughts on fructose being linked to glycation in humans? I try to avoid any concernrated forms of fructose especially HFCS, soda, fruit juice, etc... but do you think eating large amounts of fruit for years on end is likely to accelerate the aging process? (through glycation from long term exposure to fructose)? From a pure health perspective, is there anything fruit does that vegetables can't?

I think one cup of blueberries has like 7 grams of fructose
bananas have a little more
peaches and cherries are the lowest
l

suckmymuscle

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Re: The Frutarian.
« Reply #36 on: November 27, 2011, 01:15:42 PM »
no, didnt say that but not that i expect you to have critical thinking skills. Fructose which is one of the sugars in fruits (varying degrees) is not a healthy sugar.

  It is not healthy in large amounts just like glucose, protein and fatty acids are not either. All the studies done were with high fructose corn syrup. That is sucrose, a disaccharide composed of glucose and fructose. How can you isolate the fructose and say that it is the culprit? How can you know that is not the glucose? And all of the macronutrients and their component parts are unhealthy in large doses.

Quote
  There are things in fruits that are not healthy and not healthy for certain individuals. several servings of fruits a day is healthy due to the vitamins, phytochemicals etc. but having a fruit only or heavy diet is not, its just to much sugar which has all kinds of negative effects on health. Human physiology is such that meat and fatty acids are required in most cases. vegetables have more benefits then fruits while improving glucose regulation. Why do you think we have HCL in our stomachs, to digest carbs? lol.

  The hydrochloric acid in our stomachs is literally 10 times weaker than that in predators. And it is there because we need it to break down any protein - not just the one in meat. Meat is not required: amino acids are. And the essential fatty acids don't appear to be esential at all. There is evidence that the body can make both linoleic as well as alpha-linolenic acids from oleic and palmitoleic acids, which are omega-9 and omega-7 respectively and present in small amounts in most fruits and vegetables.

SUCKMYMUSCLE

flinstones1

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Re: The Frutarian.
« Reply #37 on: November 27, 2011, 01:15:50 PM »
no, didnt say that but not that i expect you to have critical thinking skills. Fructose which is one of the sugars in fruits (varying degrees) is not a healthy sugar. There are things in fruits that are not healthy and not healthy for certain individuals. several servings of fruits a day is healthy due to the vitamins, phytochemicals etc. but having a fruit only or heavy diet is not, its just to much sugar which has all kinds of negative effects on health. Human physiology is such that meat and fatty acids are required in most cases. vegetables have more benefits then fruits while improving glucose regulation. Why do you think we have HCL in our stomachs, to digest carbs? lol.

interesting. I would agree that vegetables are superior, however berries are an exception. There was a study done, showing that spinach, strawberry, and blueberry all improoved memory in mice. But only the blueberries were linked to increases in motor skills and balance/coordination. Blueberries are antiaging for the brain, and have the highest biovailibility of antioxidants of any fruit or vegetables to my knowledge. They cross the blood barrier very easily.

blueberries are antioxidants to what whey is to protein, highly biovailable. Blueberries have also been shown to improove memory, anti aging to the brain, and even increase IGF levels. Strawberries work in different parts of the brain, so a combination of berries is best IMO.

berries, green vegetables, bananas for the kidneys and watermelon for the lycopene.. alll that is needed
l

suckmymuscle

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #38 on: November 27, 2011, 01:19:47 PM »
sucky what are your thoughts on fructose being linked to glycation in humans? I try to avoid any concernrated forms of fructose especially HFCS, soda, fruit juice, etc... but do you think eating large amounts of fruit for years on end is likely to accelerate the aging process? (through glycation from long term exposure to fructose)? From a pure health perspective, is there anything fruit does that vegetables can't?

I think one cup of blueberries has like 7 grams of fructose
bananas have a little more
peaches and cherries are the lowest

  Every metabolic reaction in your body is reducing and leads to electrons being stolen from your cells by oxidation. This is inevitable. Fructose seems to increase glycation more than glucose, but glycation is more related to protein intake than sugar intake.

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Re: The Frutarian.
« Reply #39 on: November 27, 2011, 01:21:05 PM »
Been eating 6-8 servings of raw fruit and veggies for the last 15 years. That's why I look decades younger than my peers.

What's an example of what you eat in a day?

I look younger than i am but then i'm only early 30's and told a lot i look 25. At 28 i was told i was 22-3. Even though i eat enough fruit and vegies i believe it's more staying lean and the training.

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Re: The Frutarian.
« Reply #40 on: November 27, 2011, 01:22:05 PM »
no, didnt say that but not that i expect you to have critical thinking skills. Fructose which is one of the sugars in fruits (varying degrees) is not a healthy sugar. There are things in fruits that are not healthy and not healthy for certain individuals. several servings of fruits a day is healthy due to the vitamins, phytochemicals etc. but having a fruit only or heavy diet is not, its just to much sugar which has all kinds of negative effects on health. Human physiology is such that meat and fatty acids are required in most cases. vegetables have more benefits then fruits while improving glucose regulation. Why do you think we have HCL in our stomachs, to digest carbs? lol.

of course you love meat you name is Necrosis

HCL breaks down all things we injest. How come vegetarians never get ulcers an its always the meat eaters that do. That would be a meat induced HCL problem, this all leads to cancers= inflamation same as ulcers wich are an inflamation
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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #41 on: November 27, 2011, 01:22:57 PM »
no one only just eats fruits and veggies you gotta get some beans and rice in there too
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Re: The Frutarian.
« Reply #42 on: November 27, 2011, 01:53:37 PM »
  It is not healthy in large amounts just like glucose, protein and fatty acids are not either. All the studies done were with high fructose corn syrup. That is sucrose, a disaccharide composed of glucose and fructose. How can you isolate the fructose and say that it is the culprit? How can you know that is not the glucose? And all of the macronutrients and their component parts are unhealthy in large doses.

  The hydrochloric acid in our stomachs is literally 10 times weaker than that in predators. And it is there because we need it to break down any protein - not just the one in meat. Meat is not required: amino acids are. And the essential fatty acids don't appear to be esential at all. There is evidence that the body can make both linoleic as well as alpha-linolenic acids from oleic and palmitoleic acids, which are omega-9 and omega-7 respectively and present in small amounts in most fruits and vegetables.

SUCKMYMUSCLE

there are ample studies of fructose only, i was saying that the fructose only studies corroborate the HFCS studies, there are a multitude of human trials with only fructose, just pubmed it and use the limits humans. so the argument that its all hfcs is false, its not. As to how to discern the effects of glucose and fructose its impossible to do 100% however, studies have used up to 95% fructose and 5% glucose and that results continued to skew in a trend that was predicted if fructose was the culprit, its a correlation study, which lead the way for DBRPCT which corroborated the findings.

arguing against or for fruit is way harder then just railing against fructose as some fruits like bitter melon contain glycosides similar to glyburide and also has lectins which have insulin like effects, such that it is an anti-diabetic treatment. Therefore if you were to examing fructose using bitter melon and its effects on postpradial glucose and insulin secretion you would get a completely different story, here in lies the issue. Fructose is bad i have no qualms or reservations about that, the science bares this out, however, fruits is healthy for a whole other host of reasons.

are you saying the acidity of the HCL is higher in humans because its known to be between .9 and 2 which leaves little room for more acidity. Im no chemistry wiz though but are you maybe saying that the mmols of hcl is more dilute in the stomach contents? expand if you will. Also, saying its weaker or less without qualification is no argument at all, so what its more basic if this is true it doesn't take into account our elaborate enzymatic systems nor the difference between our alimentary canal and other species, its meaningless.


the conversion rates of omega-3 to eph and dha is terrible and not conducive to health, the other conversions if certain to exist are probably just as poor although i would have to see the data but some evidence as you articulated indicates that its not a well understood nor studied process, nor could it sustain life in ample amounts.The meat thing i agree, not sure if i said meat was essential, pretty sure i didnt but regardless HCL is for protein degredation and as part of the innate immune system.

HFCS is not sucrose in a traditional sense as varying levels of the constituents are availible with varying bonds that aren't found in traditional table sugar, its a manufactured sweetner with the composition of sucrose. however, that is a side bar argument.

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Re: The Frutarian.
« Reply #43 on: November 27, 2011, 01:56:50 PM »
of course you love meat you name is Necrosis

HCL breaks down all things we injest. How come vegetarians never get ulcers an its always the meat eaters that do. That would be a meat induced HCL problem, this all leads to cancers= inflamation same as ulcers wich are an inflamation

what? ulcers arent caused by HCL so there is no point in responding to this, gastrin secretion is a positive feedback loop as well.

i have no idea if ulcers are higher in vegetarians or omnivores, do you have any proof? even if you did why jump to the conclusion that meat is the problem, perhaps its the bacteria which can reside in meat that is inducing ulceration, H. PYLORI sayz OH HAI.

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #44 on: November 27, 2011, 01:57:56 PM »
no one only just eats fruits and veggies you gotta get some beans and rice in there too

You had the pathogenesis of ulcers wrong but this is 100% right.

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #45 on: November 27, 2011, 01:59:40 PM »
just to qualify, save cases like zollinger-ellison syndrome normal hcl does not induce ulcers. I can describe how they occur if you actually care.

im not against fruit ftr. I think berries are the healthiest thing you can eat.

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #46 on: November 27, 2011, 02:08:52 PM »
He has been eating nothing but fruit for 6 years. And yet, he has so much energy that he can run 4 marathons in a week, and his complexion glows with vitality. His blood work shows outstanding health: extremely low LDL cholesterol and high HDL cholesterol, extremely low levels of uric acid, amonia and creatinine in urine and the most amazing thing of all is that, even though he gets 90% of his calories from carbohydrates, he has extremely low levels of insulin. Fruit is rich in fructose, a monosaccharide that dramatically increases the body's ability to use glucose effectively. And yes, he is very skinny, but that is not due to the fruitarian diet but due to the fact that he runs 20 miles a day, every day. Meat-eating marathoners look exactly like him.

  I do not recommend this diet. Fruits are too low in EFAs and especially Calcium, Phosphorus, Iron and Zinc, and the little it does has very low bioavailability compared to the chelated minerals in animal proteins. But still, this diet is very good for the liver, kidneys, heart and vascular systems.

  


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What a tool! Steve Jobs was basically a fruitarian and look what happened to him! He eats out of the carton and then puts it back.

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #47 on: November 27, 2011, 02:21:40 PM »
interesting

necrosis backwards is sis or cen= "cease or sin"
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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #48 on: November 27, 2011, 02:33:09 PM »
Whole fruit has fibre don't forget which makes it a lot different than corn syrup. There is a video on YouTube of some guy who takes his BGL prior to eating 14 bananas at once and his levels don't rise above normal limits. I will try to find it.

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Re: The Frutarian - eat nothing but fruit for 6 years
« Reply #49 on: November 27, 2011, 02:37:00 PM »
So would anyone here use fruit for cutting?