Heart Surgeon: You’ve Been LIED TO About Cholesterol & Heart Disease

[oldtimer1]

This thread has lead me to look at a lot of stuff on the internet regarding heart disease. One conclusion I read is that statins are beneficial to people who have had cardiac trouble but have not shown to be helpful in a significant percentage to people with high cholesterol with no exiting heart disease.

 

[mryorkielover]

This guy is pretty smart on things like this.

eature=share



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[Humble Narcissist]

I only follow Dr. Fauci for medical advices. I plan on attending his class next semester

To attend his class you must have at least 4 booster shots to protect against the deadly Covid.

[Gym Rat]

I’m not sure where you’ve heard this, it’s well-established that statins reduce mortality in people at high risk for cardiovascular disease.  Posting from my phone right now, but would be happy to furnish studies—although they’re not hard to find. 

Ive read that for folks with high cardiovascular risk (small percent) they can help mortality, but the millions of users with "Little to no" risk, its not affected.
So millions are using them for nothing. Also new cases of diabetes after using goes up in a big way... (All of this may have changed in the decade since the books Ive read) of course they were from "anti statin" Dr's).

NO matter what they help (or dont  help), its a cash grab and way "over prescribed"...

[JackTheRipper]

Just wondering, how many of you experts posting links have heart disease or had a heart attack and actually use statins?

[IroNat]

Decide for yourself based on your own research whether the benefits from taking statins will outweigh the possible side effects.

Don't just blindly follow your doctor's advices.

[OAK]

Decide for yourself based on your own research whether the benefits from taking statins will outweigh the possible side effects.

Don't just blindly follow your doctor's advices.

Great idea!

What could possibly go wrong?

🙂

[loco]

IroNat,

Your peer-reviewed, double-blind, published intervention studies are no match for OAK’s memes.  Just give up already.

[IroNat]

This thread has lead me to look at a lot of stuff on the internet regarding heart disease. One conclusion I read is that statins are beneficial to people who have had cardiac trouble but have not shown to be helpful in a significant percentage to people with high cholesterol with no exiting heart disease.

Good determination.

[jwb]

The high death rates of people with Familial Hypercholesterolemia (FH) proves that very high LDL cholesterol isn’t a good thing.

Dallas Mccarver had tons of heart disease in his family and had established high cholesterol levels according to his autopsy, so he may have had FH. He had advanced atherosclerosis when he died which is very rare for someone age 26.

Rich Piana was way older, ate ice cream every night, and had very mild atherosclerosis so he definitely didn’t have FH.

FH is a genetic lipid disorder and most normal people without FH can lower their levels enough if they stop eating like pigs (saturated fat and refined carbs) and lose weight.

[loco]

The high death rates of people with Familial Hypercholesterolemia (FH) proves that very high LDL cholesterol isn’t a good thing.

Dallas Mccarver had tons of heart disease in his family and had established high cholesterol levels according to his autopsy, so he may have had FH. He had advanced atherosclerosis when he died which is very rare for someone age 26.

Rich Piana was way older, ate ice cream every night, and had very mild atherosclerosis so he definitely didn’t have FH.

FH is a genetic lipid disorder and most normal people without FH can lower their levels enough if they stop eating like pigs (saturated fat and refined carbs) and lose weight.

"Studies on the FH population, however, provide an extensive literature highlighting inconsistencies with the lipid hypothesis. For example, if LDL-C is inherently atherogenic, the burden of atherosclerosis should increase with the time of exposure to LDL-C. That is, cardiovascular mortality would be predicted to increase with age as a direct consequence of the time of exposure to LDL-C. To the contrary, CVD mortality in FH individuals declines with age [41]. Elderly individuals with FH exhibit an equivalent risk of CVD mortality to those in the non-FH population, despite a lifetime of exposure to high LDL-C. This finding directly conflicts with the dual component hypothesis that LDL-C is inherently atherogenic, and that CVD risk increases with the duration of LDL-C exposure [42]. That elderly FH individuals exposed to decades of high LDL-C demonstrate no increase in CVD mortality, as well as no increase in morbidity, for example, ischemic stroke [43], compared to the general population, undermines the lipid hypothesis, that is, that high LDL-C is inherently atherogenic.

Further challenging to the lipid hypothesis is that FH individuals have a lifetime all-cause mortality rate which is equivalent to, or even lower, than that of the general population [41,44–47]."

https://journals.lww.com/co-endocrinology/fulltext/2022/10000/statin_therapy_is_not_warranted_for_a_person_with.14.aspx

[IroNat]

KO by loco.

[jwb]

"Studies on the FH population, however, provide an extensive literature highlighting inconsistencies with the lipid hypothesis. For example, if LDL-C is inherently atherogenic, the burden of atherosclerosis should increase with the time of exposure to LDL-C. That is, cardiovascular mortality would be predicted to increase with age as a direct consequence of the time of exposure to LDL-C. To the contrary, CVD mortality in FH individuals declines with age [41]. Elderly individuals with FH exhibit an equivalent risk of CVD mortality to those in the non-FH population, despite a lifetime of exposure to high LDL-C. This finding directly conflicts with the dual component hypothesis that LDL-C is inherently atherogenic, and that CVD risk increases with the duration of LDL-C exposure [42]. That elderly FH individuals exposed to decades of high LDL-C demonstrate no increase in CVD mortality, as well as no increase in morbidity, for example, ischemic stroke [43], compared to the general population, undermines the lipid hypothesis, that is, that high LDL-C is inherently atherogenic.

Further challenging to the lipid hypothesis is that FH individuals have a lifetime all-cause mortality rate which is equivalent to, or even lower, than that of the general population [41,44–47]."

https://journals.lww.com/co-endocrinology/fulltext/2022/10000/statin_therapy_is_not_warranted_for_a_person_with.14.aspx

Effect of Long-Term Exposure to Lower Low-Density Lipoprotein Cholesterol Beginning Early in Life on the Risk of Coronary Heart Disease: A Mendelian Randomization Analysisj

Methods

In a meta-analysis combining nonoverlapping data from 312,321 participants, long-term exposure to lower LDL-C was associated with a 54.5% reduction in the risk of CHD for each mmol/l (38.7 mg/dl) lower LDL-C.

Conclusions

Prolonged exposure to lower LDL-C beginning early in life is associated with a substantially greater reduction in the risk of CHD.

……………………………….

They analyzed decades worth of blood tests of over 300,000 people and the relationship of CHD and LDL levels showed a linear relationship.

If you don’t believe this is an issue go on some FH Facebook groups and read the stories of 30/40 year olds with advanced heart disease when their LDL gets really high.

[jwb]

Reductions in all-cause, cancer, and coronary mortality in statin-treated patients with heterozygous familial hypercholesterolaemia: a prospective registry study

Aims

To examine the changes in coronary, all-cause, and cancer mortality in patients with heterozygous familial hypercholesterolaemia (FH) before and after lipid-lowering therapy with statins.

Methods and results

A total of 3382 patients (1650 men) aged <80 years were recruited from 21 lipid clinics in the United Kingdom and followed prospectively between 1980 and 2006 for 46 580 person-years. There were 370 deaths, including 190 from coronary heart disease (CHD) and 90 from cancer. The standardized mortality ratio (compared with the population in England and Wales) was calculated before and from 1 January 1992. In patients aged 20–79 years, CHD mortality fell significantly by 37% (95% CI=7–56) from 3.4- to 2.1-fold excess. Primary prevention resulted in a 48% reduction in CHD mortality from 2.0-fold excess to none, with a smaller reduction of nearly 25% in patients with established disease. Coronary mortality was reduced more in women than in men. In patients without known CHD at registration, all-cause mortality from 1992 was 33% (21–43), lower than in the general population, mainly due to a 37% (21–50) lower risk of fatal cancer.

Conclusion

The results emphasize the importance of early identification of FH and treatment with statins.

[loco]

Why aren’t you posting the links to the sources you are citing?

[OAK]

Reductions in all-cause, cancer, and coronary mortality in statin-treated patients with heterozygous familial hypercholesterolaemia: a prospective registry study

Aims

To examine the changes in coronary, all-cause, and cancer mortality in patients with heterozygous familial hypercholesterolaemia (FH) before and after lipid-lowering therapy with statins.

Methods and results

A total of 3382 patients (1650 men) aged <80 years were recruited from 21 lipid clinics in the United Kingdom and followed prospectively between 1980 and 2006 for 46 580 person-years. There were 370 deaths, including 190 from coronary heart disease (CHD) and 90 from cancer. The standardized mortality ratio (compared with the population in England and Wales) was calculated before and from 1 January 1992. In patients aged 20–79 years, CHD mortality fell significantly by 37% (95% CI=7–56) from 3.4- to 2.1-fold excess. Primary prevention resulted in a 48% reduction in CHD mortality from 2.0-fold excess to none, with a smaller reduction of nearly 25% in patients with established disease. Coronary mortality was reduced more in women than in men. In patients without known CHD at registration, all-cause mortality from 1992 was 33% (21–43), lower than in the general population, mainly due to a 37% (21–50) lower risk of fatal cancer.

Conclusion

The results emphasize the importance of early identification of FH and treatment with statins.

Game.

Set.

Match.

Thanks for posting!

 

[loco]

Game.

Set.

Match.

Thanks for posting!

 

Would have been helpful if he'd posted the links.

[Megalodon]

Anyone take vitamin K2(not to be confused with K aka K1) or get it in foods like Natto?

Vitamin K2 allegedly helps prevent arterial calcification and regulates calcium metabolism so it's properly deposited in bones and teeth.

[loco]

Anyone take vitamin K2(not to be confused with K aka K1) or get it in foods like Natto?

Vitamin K2 allegedly helps prevent arterial calcification and regulates calcium metabolism so it's properly deposited in bones and teeth.

If you supplement vitamin D or consume a lot of products fortified with vitamin D, which significantly increases calcium absorption, then yes, you should be supplementing vitamin K with your vitamin D for the reasons you mentioned.

If you are not obese and eat a high fat (vitamin D is fat soluble), low carb, whole foods, animal-based diet, then you don't need to supplement vitamin D or vitamin K, unless your vitamin D levels are low for some strange reason which should be looked at by a medical professional.

[Rambone]

Anyone take vitamin K2(not to be confused with K aka K1) or get it in foods like Natto?

Vitamin K2 allegedly helps prevent arterial calcification and regulates calcium metabolism so it's properly deposited in bones and teeth.

I do. D3, K2, CoQ10 and an absorbable form of magnesium seems useful but what the fuck do I know? I’m just a former sheet metal worker living with my parents and staying up all night reading the muscle books

[Gym Rat]

Anyone take vitamin K2(not to be confused with K aka K1) or get it in foods like Natto?

Vitamin K2 allegedly helps prevent arterial calcification and regulates calcium metabolism so it's properly deposited in bones and teeth.

I take Natto (12,000 FU) (at night as it makes me lethargic), Magnesium, D3-K2 (MK7), Real Red Yeast Rice (w/ Monacolin-K which is natural Lovastatin from the rice fermentation(Bought from other countries where its legal), CO-Q10, Niacin...

[mryorkielover]

If you supplement vitamin D or consume a lot of products fortified with vitamin D, which significantly increases calcium absorption, then yes, you should be supplementing vitamin K with your vitamin D for the reasons you mentioned.

If you are not obese and eat a high fat (vitamin D is fat soluble), low carb, whole foods, animal-based diet, then you don't need to supplement vitamin D or vitamin K, unless your vitamin D levels are low for some strange reason which should be looked at by a medical professional.

I would agree with you if you knew you were getting Quality food. In this day and age, that is not a given. If you buy 2 apples from different places, they will be different. One apple might have more nutrients than the other, or it might have more pesticides. Regardless if it says organic. Alot of lying to the public and the FDA not really protecting the consumer. That is why I would ALWAYS take supplements, regardless of how clean and healthy you think your diet is. Supplements fill in the gaps of what your diet might be missing and now a days, it's a lot.

[joswift]

I would agree with you if you knew you were getting Quality food. In this day and age, that is not a given. If you buy 2 apples from different places, they will be different. One apple might have more nutrients than the other, or it might have more pesticides. Regardless if it says organic. Alot of lying to the public and the FDA not really protecting the consumer. That is why I would ALWAYS take supplements, regardless of how clean and healthy you think your diet is. Supplements fill in the gaps of what your diet might be missing and now a days, it's a lot.

even the amount of calories you eat is a rough guess

Take 10 Ribeye Steak, depending on its fat/lean meat ratio it could vary by 100 cals minimum

[Dokey111]

You need to stop thinking anybody knows anything about anything.

[mryorkielover]

Anyone take vitamin K2(not to be confused with K aka K1) or get it in foods like Natto?

Vitamin K2 allegedly helps prevent arterial calcification and regulates calcium metabolism so it's properly deposited in bones and teeth.

Life Extension brand puts out a Super K product that is decent price and quality. It has 1500mcg of K1, 1000mcg of K2 as menaquinone-4, and 100 mcg of K2 as trans menaquinone-7. Seemed to be one of the best products on the market when I was looking for a K2 supplement. $19 for 90 pills. Says take 1 a day but I noticed that supplements work better when you find out how much YOUR body really needs. 1 pill might work for me but you might need 2 or more a day depending on your age and health. Not everyone is the same.