These threads keep coming up because people don't like the answer.
Not talking opinion here, simply what the drugs do. The growth isn't limited to 'work-out' muscles. Even if it were, GI muscles are almost always moving. GH also causes growth in small bones. Comparing them to regular fatasses doesn't make sense because type II diabetics have decreased insulin sensitivity. There are 4 or 5 glucose receptors in humans, why wouldn't there be more than one for insulin?
Large amounts of food obviously cause the GI tract to work harder but you don't see huge guts in naturals.
Growth in small bones? I'm not sure what you're talking about here and how it relates to this topic. Same with the 4 or 5 glucose receptors thing.
Regarding the growth of intestines etc it hasn't been quantified - if it occurs and to what degree. If you look at the HIV hGH studies they posed the possibility of organ growth accounting for some of the lbm gain. But it hasn't been looked at more closely to my knowledge. I think most of the lbm gain in those studies is simply water retention. How much the organs grow is unknown. Disgusted believes in the VAT theory and doesn't believe the organs grow since it would cause the belly to look unsymmetrical in his opinion.
Regarding VAT, hGH seems to cause reduction of it despite causing insulin resistance and consequent hyperinsulinemia. timfogarty says the guts came down in HIV patients when they dropped the growth so in those cases the distention must logically be due to something other than VAT.
And yes, I think bodybuilders on lots of growth are often severely insulin resistant, to the point of type II diabetes. Why do you think many of them say they
must use extra insulin when on lots of GH (due to elevated BG)?