The only thing that is not present is a warning...
Remember, Ephedrine is a powerful drug. It is not classed as addictive, but trust me it can be. Several years ago (over 7) I tried an ECA stack for the first time. At the time no one had worked out the idea of being off 1-2 days per week and not using for longer than 4 weeks without time off, etc...
Anyway, I built up a tolerance to Ephedrine and to Caffeine. After 3 months of continuous use (abuse), I was taking over 1000mg of Caffeine per day and about 400mg of Ephedrine per day... I never took a break... and there were days when I exceeded those amounts...
After over a year I tried to taper off the amounts but it was no use... without enough caffeine I had huge headaches and I found that without the ephedrine I couldn't concentrate... The end result was that I went cold turkey... NOT SMART... I crashed... I slept at least 12 hours per day for the next 3 months... and it took over a year for my body to recover...
ECA is an amazing fat burning stack, but abusing it is dangerous and can cause serious issues... as I stated this was years ago for me and I have used the stack since with great success... please read the advice on getbig and other sights and don't abuse it.
Cheers!
Wooo
No, it is not. Among catecholaminergic monoamines, ephedrine is one of the safest. It's sympathomimetic effects are four times less potent than that of dexamphetamine, and roughly sixteen times lower than dextro-methamhetamine hydrochloride.
There is no evidence that ephedrine raises systolic blood pressure considerably, nor that it induces vasocompression of capilaries on the central and peripheral nervous systems. It does not seem to increase intra-cranial pressure either. The metbolism of ephedrine is slow, and there lies the danger, as the curve to peak maximum lvel, of absorption, is much fster than the clearance rate, thus potentially raising it's plasma concentration to dangerous levels.
Ephedrine is biochemically and physiologically analagous to norepinephrine, a neuro-hormone used, by peripheral nerve endings, to signal vsocompression and contraction of actin myofibrils, by promoting the excretion of Calcium Ions from the peripherl neuronal dendrites and myofibril sarcoplasms. It differs structurally from more potent sympathomimetic amines, like the amphetamines, by having an Oxygen atom attached to the Carbon on it's second chiral center. This increases it's activation of beta-receptors, while conversely decreasing it's monoaminergic and especially dopaminergic effects.
By reducing ephedrine, where the Oxygen atom is replaced by one of Carbon, the new compound, desoxyephedrine(reduced ephedrine), comercially known as methamphetamine, has it's adrenergic effects increased several times - because the smaller molecule enters the alpha cells, of the adrenal glans, far more quickly, - and gains especificity to the D2DR dopaminergic receptors in neurons of both substantia nigra and pre-frontal cortex of the brain. This dramatically increases both the amplification of systolic and diastolic blood pressure and potential addictivness of the modified amine, while significantly decreases it's thermogenic and especially lipogenic effect.
When used in combination with caffeine, or other methylxantines, the lipogenic activity of alcoholic monoamines, like ephedrine, is dramatically enhanced, because methylxantines have the capacity to inhibit adenosine, which, once inhibited, blocks the activity of several prostaglandines which deactivate beta-receptors. Thus, the combination of methylxantines and alcoholic monoaminergic sympathomimetics, such as ephedrine, pseudoephedrine, phentermine, phenmetrazine or phendimetrazine, is synergistic. There is no evidence that methylxantines significantly increase the vasopressive effect and/or systolic blood pressure enhancing activity of monominergic sympathomimetics with weak noradrenergic action, such as ephedrine. In conclusion: the combination of ephedrine and caffeine is safe for most people, most of the time. Regards.
SUCKMYMUSCLE
